Rheumatoid factor, complement, and mixed cryoglobulinemia. - Wikidata - awgldk - TriplyDB

Rheumatoid factor, complement, and mixed cryoglobulinemia.

Rheumatoid factor, complement, and mixed cryoglobulinemia.

http://www.wikidata.org/entity/Q38041549

about

Role of Natural Autoantibodies and Natural IgM Anti-Leucocyte Autoantibodies in Health and Disease The expanding spectrum of HCV-related cryoglobulinemic vasculitis: a narrative review.Vasculitis with renal involvement in essential mixed cryoglobulinemia: Case report and mini-review Hepatitis C virus syndrome: A constellation of organ- and non-organ specific autoimmune disorders, B-cell non-Hodgkin's lymphoma, and cancer Molecular signatures of hepatitis C virus (HCV)-induced type II mixed cryoglobulinemia (MCII).Complement C3c as a biomarker in heart failure.Utility of immunologic testing in suspected rheumatologic disease.Vasculitis: determinants of disease patterns.Role of Natural IgM Autoantibodies (IgM-NAA) and IgM Anti-Leukocyte Antibodies (IgM-ALA) in Regulating Inflammation.The complement and immunoglobulin levels in NMO patients.Isolation, characterization, and molecular modeling of a rheumatoid factor from a Hepatitis C virus infected patient with Sjögren's syndrome.The Complement System and C1q in Chronic Hepatitis C Virus Infection and Mixed Cryoglobulinemia.

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P2860

Rheumatoid factor, complement, and mixed cryoglobulinemia.

http://www.wikidata.org/entity/Q38041549

description

article científic

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article scientifique

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articol științific

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artikel ilmiah

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artículo científico

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name

Rheumatoid factor, complement, and mixed cryoglobulinemia.

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type

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Rheumatoid factor, complement, and mixed cryoglobulinemia.

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Rheumatoid factor, complement, and mixed cryoglobulinemia.

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Clinical and Developmental Immunology

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Rheumatoid factor, complement, and mixed cryoglobulinemia.

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Peter D Gorevic

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Creative Commons Attribution 3.0 Unported

P2860

Interaction between complement receptor gC1qR and hepatitis C virus core protein inhibits T-lymphocyte proliferation.

Anti-C1q antibodies in hepatitis C virus infection

Hepatitis C virus (HCV)-induced IgG-IgM rheumatoid factor (RF) complex may be the main causal factor for cold-dependent activation of complement in patients with rheumatic disease

Role of the Receptor for the Globular Domain of C1q Protein in the Pathogenesis of Hepatitis C Virus-Related Cryoglobulin Vascular Damage

Cryoimmunoglobulinemia in rheumatoid arthritis. Significance in serum of patients with rheumatoid vasculitis.

Transcriptional repression of C4 complement by hepatitis C virus proteins.

Regulation of circulating immune complexes by complement receptor type 1 on erythrocytes in chronic viral liver diseases.

Hepatitis C virus proteins inhibit C3 complement production

Transcriptional control of genes for soluble complement cascade regulatory proteins.

Dynamic control of the complement system by modulated expression of regulatory proteins.

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439018

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scholarly article

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10.1155/2012/439018

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2012

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2012-08-26T00:00:00Z

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230812147

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cryoglobulinemia

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