Neurological perspectives on voltage-gated sodium channels. - Wikidata - awgldk - TriplyDB

Neurological perspectives on voltage-gated sodium channels.

Neurological perspectives on voltage-gated sodium channels.

http://www.wikidata.org/entity/Q38042001

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Pharmacological Alternatives for the Treatment of Neurodegenerative Disorders: Wasp and Bee Venoms and Their Components as New Neuroactive Tools Node of Ranvier disruption as a cause of neurological diseases Prolactin receptor in regulation of neuronal excitability and channels Exaggerated Nighttime Sleep and Defective Sleep Homeostasis in a Drosophila Knock-In Model of Human Epilepsy Developmental Profile of Ion Channel Specializations in the Avian Nucleus Magnocellularis Current Status of the New Antiepileptic Drugs in Chronic Pain A monoclonal antibody that targets a NaV1.7 channel voltage sensor for pain and itch relief Endogenous opioids contribute to insensitivity to pain in humans and mice lacking sodium channel Nav1.7 Nav1.7 and other voltage-gated sodium channels as drug targets for pain relief Major channels involved in neuropsychiatric disorders and therapeutic perspectives A de novo gain-of-function mutation in SCN11A causes loss of pain perception Protein interaction networks reveal novel autism risk genes within GWAS statistical noise Increased excitability and excitatory synaptic transmission during in vitro ischemia in the neonatal mouse hippocampus.Folding similarity of the outer pore region in prokaryotic and eukaryotic sodium channels revealed by docking of conotoxins GIIIA, PIIIA, and KIIIA in a NavAb-based model of Nav1.4.Pain vulnerability: a neurobiological perspective.Physiological bases of the K+ and the glutamate/GABA hypotheses of epilepsy.The sorting receptor Rer1 controls Purkinje cell function via voltage gated sodium channels.Roles of ASIC3, TRPV1, and NaV1.8 in the transition from acute to chronic pain in a mouse model of fibromyalgia.Sodium channel SCN8A (Nav1.6): properties and de novo mutations in epileptic encephalopathy and intellectual disability.Defining the nociceptor transcriptome.Transcranial application of near-infrared low-level laser can modulate cortical excitability.Update on laser-evoked potential findings in fibromyalgia patients in light of clinical and skin biopsy features.Clinical utility of eslicarbazepine: current evidence.Screening for voltage-gated sodium channel interacting peptides.Effects of moderate static magnetic fields on the voltage-gated sodium and calcium channel currents in trigeminal ganglion neurons.Oral administration of PF-01247324, a subtype-selective Nav1.8 blocker, reverses cerebellar deficits in a mouse model of multiple sclerosis Eslicarbazepine acetate for the treatment of focal epilepsy: an update on its proposed mechanisms of action.Novel sodium channel antagonists in the treatment of neuropathic pain.MicroRNA-9 induces defective trafficking of Nav1.1 and Nav1.2 by targeting Navβ2 protein coding region in rat with chronic brain hypoperfusion New Role for FDA-Approved Drugs in Combating Antibiotic-Resistant Bacteria Synthesis and biological evaluation of palmyrolide A macrocycles as sodium channel blockers towards neuroprotection.Pharmacogenetics of analgesic drugs Ultrastructural anatomy of nodes of Ranvier in the peripheral nervous system as revealed by STED microscopy.The AMPK Activator A769662 Blocks Voltage-Gated Sodium Channels: Discovery of a Novel Pharmacophore with Potential Utility for Analgesic Development.A Common Polymorphism in SCN2A Predicts General Cognitive Ability through Effects on PFC Physiology WeGET: predicting new genes for molecular systems by weighted co-expression.Characterization of Endogenous Sodium Channels in the ND7-23 Neuroblastoma Cell Line: Implications for Use as a Heterologous Ion Channel Expression System Suitable for Automated Patch Clamp Screening Peripheral nerve injury is accompanied by chronic transcriptome-wide changes in the mouse prefrontal cortex.Resveratrol attenuates cortical neuron activity: roles of large conductance calcium-activated potassium channels and voltage-gated sodium channels.Mutant cycle analysis with modified saxitoxins reveals specific interactions critical to attaining high-affinity inhibition of hNaV1.7.

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Neurological perspectives on voltage-gated sodium channels.

http://www.wikidata.org/entity/Q38042001

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Neurological perspectives on voltage-gated sodium channels.

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Neurological perspectives on voltage-gated sodium channels.

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Neurological perspectives on voltage-gated sodium channels.

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Neurological perspectives on voltage-gated sodium channels.

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Neurological perspectives on voltage-gated sodium channels.

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Neurological perspectives on voltage-gated sodium channels.

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Neurological perspectives on voltage-gated sodium channels

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François Rugiero

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John N Wood

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Mark D Baker

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Michael S Minett

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Roman Cregg

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Creative Commons Attribution-NonCommercial 3.0 Unported

P2860

Mutations of SCN1A, encoding a neuronal sodium channel, in two families with GEFS+2

X-ray structure of a voltage-dependent K+ channel

Carbamazepine for acute and chronic pain in adults

Systemic administration of local anesthetic agents to relieve neuropathic pain

Loss-of-function mutation of the SCN3B-encoded sodium channel {beta}3 subunit associated with a case of idiopathic ventricular fibrillation

Sodium channel Scn1b null mice exhibit prolonged QT and RR intervals

Genetic variation in SCN10A influences cardiac conduction

Sudden infant death syndrome-associated mutations in the sodium channel beta subunits

Sodium channel β1 subunit mutations associated with Brugada syndrome and cardiac conduction disease in humans

SCN4B-encoded sodium channel beta4 subunit in congenital long-QT syndrome

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2585-2612

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scholarly article

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10.1093/BRAIN/AWS225

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Pt 9

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135

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Niels Eijkelkamp

Robert Werdehausen

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2012-09-01T00:00:00Z

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230826275

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22961543

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transport protein

membrane proteins

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3437034

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