The role of reactive oxygen species and proinflammatory cytokines in type 1 diabetes pathogenesis.
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Suppressors of Cytokine Signaling in Sickness and in Health of Pancreatic β-CellsNeutrophils in type 1 diabetesDiabetes mellitus related bone metabolism and periodontal diseasePreferential uptake of antioxidant carbon nanoparticles by T lymphocytes for immunomodulation.Growth Hormone-Releasing Hormone in DiabetesAntagonism of the Prokineticin System Prevents and Reverses Allodynia and Inflammation in a Mouse Model of DiabetesRole of Pro-Inflammatory Cytokines and Biochemical Markers in the Pathogenesis of Type 1 Diabetes: Correlation with Age and Glycemic Condition in Diabetic Human SubjectsDeoxyhypusine synthase promotes differentiation and proliferation of T helper type 1 (Th1) cells in autoimmune diabetesImpact of Pancreatic Rat Islet Density on Cell Survival during Hypoxia.Reactive oxygen species are required for driving efficient and sustained aerobic glycolysis during CD4+ T cell activation.Evaluation of oxidative stress biomarkers in patients with fixed orthodontic appliancesRegulation of iNOS gene transcription by IL-1β and IFN-γ requires a coactivator exchange mechanism.Nitric oxide induces ataxia telangiectasia mutated (ATM) protein-dependent γH2AX protein formation in pancreatic β cells.Biotin uptake by mouse and human pancreatic beta cells/islets: a regulated, lipopolysaccharide-sensitive carrier-mediated process.Evidence of contribution of iPLA2β-mediated events during islet β-cell apoptosis due to proinflammatory cytokines suggests a role for iPLA2β in T1D development.Transcription of the gene encoding TNF-α is increased by IL-1β in rat and human islets and β-cell linesIncreased immune cell infiltration of the exocrine pancreas: a possible contribution to the pathogenesis of type 1 diabetes.Lessons from type 1 diabetes for understanding natural history and prevention of autoimmune disease.Inhibition of Ca2+-independent phospholipase A2β (iPLA2β) ameliorates islet infiltration and incidence of diabetes in NOD mice.Sustained NF-κB activation and inhibition in β-cells have minimal effects on function and islet transplant outcomesHydrogen-bonded multilayers of tannic acid as mediators of T-cell immunity.Type 1 diabetes: A predictable disease.Thiobenzothiazole-modified Hydrocortisones Display Anti-inflammatory Activity with Reduced Impact on Islet β-Cell Function.A Combination of Mitochondrial Oxidative Stress and Excess Fat/Calorie Intake Accelerates Steatohepatitis by Enhancing Hepatic CC Chemokine Production in Mice.Allium hookeri root protects oxidative stress-induced inflammatory responses and β-cell damage in pancreas of streptozotocin-induced diabetic ratsCalcium-independent phospholipases A2 and their roles in biological processes and diseases.Minireview: Directed Differentiation and Encapsulation of Islet β-Cells-Recent Advances and Future Considerations.Distinct differences in the responses of the human pancreatic β-cell line EndoC-βH1 and human islets to proinflammatory cytokines.Osteoprotegerin and Denosumab Stimulate Human Beta Cell Proliferation through Inhibition of the Receptor Activator of NF-κB Ligand Pathway.Cellular stress and innate inflammation in organ-specific autoimmunity: lessons learned from vitiligo.Low Level Pro-inflammatory Cytokines Decrease Connexin36 Gap Junction Coupling in Mouse and Human Islets through Nitric Oxide-mediated Protein Kinase Cδ.Factors Associated with Higher Pro-Inflammatory Tumor Necrosis Factor-α Levels in Young Women with Type 1 Diabetes.Maternal Obesity Promotes Diabetic Nephropathy in Rodent OffspringFeatured Article: Oxidative stress status and liver tissue defenses in diabetic rats during intensive subcutaneous insulin therapy.Nitric Oxide Suppresses β-Cell Apoptosis by Inhibiting the DNA Damage ResponseCarbonyl stress in aging process: role of vitamins and phytochemicals as redox regulatorsDespite Increased Type 1 IFN, Autoimmune Nonobese Diabetic Mice Display Impaired Dendritic Cell Response to CpG and Decreased Nuclear Localization of IFN-Activated STAT1.Do β-cells generate peroxynitrite in response to cytokine treatment?Protective effects of polyamine depletion in mouse models of type 1 diabetes: implications for therapy.NF-κB and STAT1 control CXCL1 and CXCL2 gene transcription.
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The role of reactive oxygen species and proinflammatory cytokines in type 1 diabetes pathogenesis.
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
@sq
artículo científico
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name
The role of reactive oxygen sp ...... type 1 diabetes pathogenesis.
@en
type
label
The role of reactive oxygen sp ...... type 1 diabetes pathogenesis.
@en
prefLabel
The role of reactive oxygen sp ...... type 1 diabetes pathogenesis.
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P2093
P2860
P921
P1476
The role of reactive oxygen sp ...... n type 1 diabetes pathogenesis
@en
P2093
Hubert M Tse
John A Corbett
Lindsey E Padgett
Polly A Hansen
P2860
P356
10.1111/J.1749-6632.2012.06826.X
P407
P5008
P577
2013-01-16T00:00:00Z