The utility of paradoxical components in biological circuits.
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Inhibitory signalling to the Arp2/3 complex steers cell migrationApplications of targeted proteomics in systems biology and translational medicineLife rhythm as a symphony of oscillatory patterns: electromagnetic energy and sound vibration modulates gene expression for biological signaling and healing.Connecting Neural Codes with Behavior in the Auditory System of DrosophilaEnzyme Sequestration as a Tuning Point in Controlling Response Dynamics of Signalling NetworksA design principle underlying the paradoxical roles of E3 ubiquitin ligasesSynthetic circuit for exact adaptation and fold-change detectionA fundamental trade-off in covalent switching and its circumvention by enzyme bifunctionality in glucose homeostasisA unified design space of synthetic stripe-forming networks.Robust network structure of the Sln1-Ypd1-Ssk1 three-component phospho-relay prevents unintended activation of the HOG MAPK pathway in Saccharomyces cerevisiaeProcessing Oscillatory Signals by Incoherent Feedforward Loops.A Regulatory Circuit Composed of a Transcription Factor, IscR, and a Regulatory RNA, RyhB, Controls Fe-S Cluster Delivery.Cycling Transcriptional Networks Optimize Energy Utilization on a Genome Scale.Alternate wiring of a KNOXI genetic network underlies differences in leaf development of A. thaliana and C. hirsuta.Neural fate decisions mediated by combinatorial regulation of Hes1 and miR-9Unravelling druggable signalling networks that control F508del-CFTR proteostasisA cellular and regulatory map of the cholinergic nervous system of C. elegans.Differential Masking of Natural Genetic Variation by miR-9a in Drosophila.Identification of Unintuitive Features of Sumoylation through Mathematical Modeling.Enhanced fasting glucose turnover in mice with disrupted action of TUG protein in skeletal muscle.The DNA damage response induces inflammation and senescence by inhibiting autophagy of GATA4.Paradoxical signaling regulates structural plasticity in dendritic spinesCyclic di-GMP mediates a histidine kinase/phosphatase switch by noncovalent domain cross-linkingInnate immune memory and homeostasis may be conferred through crosstalk between the TLR3 and TLR7 pathwaysYeast GPCR signaling reflects the fraction of occupied receptors, not the numberA proteolytic pathway that controls glucose uptake in fat and muscle.Pervasive robustness in biological systems.Local sampling paints a global picture: Local concentration measurements sense direction in complex chemical gradients.The Arp2/3 inhibitory protein arpin induces cell turning by pausing cell migration.The Paradox of p53: What, How, and Why?Weak-binding molecules are not drugs?-toward a systematic strategy for finding effective weak-binding drugs.SUMO and the robustness of cancer.Synthetic Immunology: Hacking Immune Cells to Expand Their Therapeutic Capabilities.Ubiquitin-Dependent Regulation of Stem Cell Biology.Cellular sensory mechanisms for detecting specific fold-changes in extracellular cues.Mechanical stretch triggers rapid epithelial cell division through Piezo1.Secreting and sensing the same molecule allows cells to achieve versatile social behaviorsSF3B1 is a stress-sensitive splicing factor that regulates both HSF1 concentration and activity.Transcriptional control of quorum sensing and associated metabolic interactions in Pseudomonas syringae strain B728a.Quantitative assessment of cell fate decision between autophagy and apoptosis.
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P2860
The utility of paradoxical components in biological circuits.
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
@sq
artículo científico
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name
The utility of paradoxical components in biological circuits.
@en
type
label
The utility of paradoxical components in biological circuits.
@en
prefLabel
The utility of paradoxical components in biological circuits.
@en
P1433
P1476
The utility of paradoxical components in biological circuits.
@en
P2093
P304
P356
10.1016/J.MOLCEL.2013.01.004
P577
2013-01-01T00:00:00Z