Role of PI3K/Akt signaling in memory CD8 T cell differentiation.
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IL-15-PI3K-AKT-mTOR: A Critical Pathway in the Life Journey of Natural Killer CellsCD73 and CD39 ectonucleotidases in T cell differentiation: Beyond immunosuppressionBeauvericin ameliorates experimental colitis by inhibiting activated T cells via downregulation of the PI3K/Akt signaling pathwayDo PI3-kinase mutations drive T cells insane?Dominant-activating germline mutations in the gene encoding the PI(3)K catalytic subunit p110δ result in T cell senescence and human immunodeficiencyInhibition of Akt signaling promotes the generation of superior tumor-reactive T cells for adoptive immunotherapy.MicroRNA-17 inhibits tumor growth by stimulating T-cell mediated host immune response.Akt inhibition enhances expansion of potent tumor-specific lymphocytes with memory cell characteristics.Intrinsic MyD88-Akt1-mTOR Signaling Coordinates Disparate Tc17 and Tc1 Responses during Vaccine Immunity against Fungal Pneumonia.CD8low CD100- T Cells Identify a Novel CD8 T Cell Subset Associated with Viral Control during Human Hantaan Virus Infection.Identification of novel microRNA signatures linked to acquired aplastic anemia.Phosphatidylinositol 3-Kinase p110δ Isoform Regulates CD8+ T Cell Responses during Acute Viral and Intracellular Bacterial InfectionsAsymptomatic memory CD8+ T cells: from development and regulation to consideration for human vaccines and immunotherapeuticsPolyfunctional Melan-A-specific tumor-reactive CD8(+) T cells elicited by dacarbazine treatment before peptide-vaccination depends on AKT activation sustained by ICOS.miR-125b regulates differentiation and metabolic reprogramming of T cell acute lymphoblastic leukemia by directly targeting A20.Ex vivo Akt inhibition promotes the generation of potent CD19CAR T cells for adoptive immunotherapy.mTORC1 regulates mannose-6-phosphate receptor transport and T-cell vulnerability to regulatory T cells by controlling kinesin KIF13A.NKG2D stimulation of CD8+ T cells during priming promotes their capacity to produce cytokines in response to viral infection in mice.Akt1 and -2 inhibition diminishes terminal differentiation and enhances central memory CD8(+) T-cell proliferation and survival.Tissue-resident T cells lose their S1P1 exit visasKeeping memories aliveRegulation of T cell alloimmunity by PI3Kγ and PI3Kδ.Ezh2 phosphorylation state determines its capacity to maintain CD8+ T memory precursors for antitumor immunity.The sorafenib anti-relapse effect after alloHSCT is associated with heightened alloreactivity and accumulation of CD8+PD-1+ (CD279+) lymphocytes in marrow.Incomplete Memories: The Natural Suppression of Tissue-Resident Memory CD8 T Cells in the Lung.PI3K orchestration of the in vivo persistence of chimeric antigen receptor-modified T cells.The Respiratory Environment Diverts the Development of Antiviral Memory CD8 T Cells.A PI3K p110α-selective inhibitor enhances the efficacy of anti-HER2/neu antibody therapy against breast cancer in mice.AKT-inhibition facilitates generation of polyfunctional stem cell memory-like CD8 T cells for adoptive immunotherapyClass-Switch Recombination (CSR)/Hyper-IgM (HIGM) Syndromes and Phosphoinositide 3-Kinase (PI3K) Defects
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Role of PI3K/Akt signaling in memory CD8 T cell differentiation.
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article científic
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article scientifique
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articol științific
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articolo scientifico
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artigo científico
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artigo científico
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artículo científico
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Role of PI3K/Akt signaling in memory CD8 T cell differentiation.
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Role of PI3K/Akt signaling in memory CD8 T cell differentiation.
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prefLabel
Role of PI3K/Akt signaling in memory CD8 T cell differentiation.
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P2860
P356
P1476
Role of PI3K/Akt signaling in memory CD8 T cell differentiation.
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P2093
P2860
P356
10.3389/FIMMU.2013.00020
P577
2013-02-01T00:00:00Z