Animal models of hyperfunctioning parathyroid diseases for drug development.

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Parathyroid diseases and animal models.Cinacalcet HCl suppresses Cyclin D1 oncogene-derived parathyroid cell proliferation in a murine model for primary hyperparathyroidism.Matrix extracellular phosphoglycoprotein is expressed in causative tumors of oncogenic osteomalacia.

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Animal models of hyperfunctioning parathyroid diseases for drug development.

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http://www.wikidata.org/entity/Q38089204

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Animal models of hyperfunctioning parathyroid diseases for drug development.

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Animal models of hyperfunctioning parathyroid diseases for drug development.

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Animal models of hyperfunctioning parathyroid diseases for drug development.

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Animal models of hyperfunctioning parathyroid diseases for drug development.

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Masaaki Inaba

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Takehisa Kawata

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Yasuo Imanishi

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Yoshiki Nishizawa

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Menin, the product of the MEN1 gene, is a nuclear protein

Autosomal dominant hypocalcaemia caused by a Ca(2+)-sensing receptor gene mutation

Mutations in the human Ca(2+)-sensing receptor gene cause familial hypocalciuric hypercalcemia and neonatal severe hyperparathyroidism

Mutation and cancer: statistical study of retinoblastoma

HRPT2 mutations are associated with malignancy in sporadic parathyroid tumours

Definition, evaluation, and classification of renal osteodystrophy: a position statement from Kidney Disease: Improving Global Outcomes (KDIGO)

A novel cyclin encoded by a bcl1-linked candidate oncogene

Human D-type cyclin

Physical interaction of the retinoblastoma protein with human D cyclins

Positional cloning of the gene for multiple endocrine neoplasia-type 1

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727-740

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scholarly article

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10.1517/17460440903022743

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English

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2009-07-01T00:00:00Z

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23489166

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Animal models of hyperfunctioning parathyroid diseases for drug development.

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P2860

P2860

Animal models of hyperfunctioning parathyroid diseases for drug development.

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type

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P1476

P2093

P2860

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P407

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P698

P356

P1433

P1476

P2093

P2860

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P356

P407

P433

P478

P577

P698