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Role of ROS and RNS Sources in Physiological and Pathological ConditionsCompromised redox homeostasis, altered nitroso-redox balance, and therapeutic possibilities in atrial fibrillationThe Role of Oxidative Stress in Neurodegenerative DiseasesExploiting the Pleiotropic Antioxidant Effects of Established Drugs in Cardiovascular DiseaseMitochondrial dysfunction and tissue injury by alcohol, high fat, nonalcoholic substances and pathological conditions through post-translational protein modificationsOxidative Stress and the Use of Antioxidants in StrokeAnti-Oxidants in Parkinson's Disease Therapy: A Critical Point of ViewProtection of Bovine Mammary Epithelial Cells from Hydrogen Peroxide-Induced Oxidative Cell Damage by ResveratrolAntioxidants in Translational MedicineMitochondrial ROS Metabolism: 10 Years Later.Role of CYP2E1 in mitochondrial dysfunction and hepatic tissue injury in alcoholic and non-alcoholic diseases.Time-course and intensity-based classifications of oxidative stresses and their potential application in biomedical, comparative and environmental research.Mitochondria-Targeted Triphenylphosphonium-Based Compounds: Syntheses, Mechanisms of Action, and Therapeutic and Diagnostic Applications.Mitochondrial dysfunction and defects in lipid homeostasis as therapeutic targets in neurodegeneration with brain iron accumulation.Oxidative Stress in Neurodegenerative Diseases: From Molecular Mechanisms to Clinical ApplicationsLinking an α-tocopherol derivative to cobalt(0) nanomagnets: magnetically responsive antioxidants with superior radical trapping activity and reduced cytotoxicity.Exercise redox biochemistry: Conceptual, methodological and technical recommendations.Synthesis of a Functionalized Benzofuran as a Synthon for Salvianolic Acid C Analogues as Potential LDL Antioxidants.Antioxidant properties in a non-polar environment of difluoromethyl bioisosteres of methyl hydroxycinnamates.Identification of Non-Electrophilic Nrf2 Activators from Approved Drugs.13 reasons why the brain is susceptible to oxidative stress.Mitochondrial ROS cause motor deficits induced by synaptic inactivity: Implications for synapse pruning.Profiling donepezil template into multipotent hybrids with antioxidant properties.New Therapies for Asthma and Chronic Obstructive Pulmonary Disease.Role of ROS and Nutritional Antioxidants in Human Diseases.Strategies to decrease oxidative stress biomarker levels in human medical conditions: A meta-analysis on 8-iso-prostaglandin F2α.
P2860
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P2860
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
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artículo científico
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name
Antioxidants as therapies: can we improve on nature?
@en
type
label
Antioxidants as therapies: can we improve on nature?
@en
prefLabel
Antioxidants as therapies: can we improve on nature?
@en
P1476
Antioxidants as therapies: can we improve on nature?
@en
P2093
Michael P Murphy
P356
10.1016/J.FREERADBIOMED.2013.04.010
P577
2013-04-18T00:00:00Z