The pathobiology of the oncogenic tyrosine kinase NPM-ALK: a brief update.
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Reversal of microRNA-150 silencing disadvantages crizotinib-resistant NPM-ALK(+) cell growthSensitivity Analysis of the NPM-ALK Signalling Network Reveals Important Pathways for Anaplastic Large Cell Lymphoma Combination Therapy.STAT3 in Cancer-Friend or Foe?Crosstalk between microRNA and DNA Methylation Offers Potential Biomarkers and Targeted Therapies in ALK-Positive LymphomasSilibinin suppresses NPM-ALK, potently induces apoptosis and enhances chemosensitivity in ALK-positive anaplastic large cell lymphoma.The use of cellular thermal shift assay (CETSA) to study Crizotinib resistance in ALK-expressing human cancers.High expression of β-catenin contributes to the crizotinib resistant phenotype in the stem-like cell population in neuroblastoma.Novel Molecular Challenges in Targeting Anaplastic Lymphoma Kinase in ALK-Expressing Human Cancers.The absence of a novel intron 19-retaining ALK transcript (ALK-I19) and MYCN amplification correlates with an excellent clinical outcome in neuroblastoma patients.The Pathological Spectrum of Systemic Anaplastic Large Cell Lymphoma (ALCL).Refractory ALK-Positive Anaplastic Large Cell Lymphoma: Long Term Survival with Crizotinib
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The pathobiology of the oncogenic tyrosine kinase NPM-ALK: a brief update.
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article científic
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article scientifique
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articol științific
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articolo scientifico
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artigo científico
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artigo científico
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artigo científico
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artículo científico
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name
The pathobiology of the oncogenic tyrosine kinase NPM-ALK: a brief update.
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The pathobiology of the oncogenic tyrosine kinase NPM-ALK: a brief update.
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prefLabel
The pathobiology of the oncogenic tyrosine kinase NPM-ALK: a brief update.
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P2860
P356
P1476
The pathobiology of the oncogenic tyrosine kinase NPM-ALK: a brief update.
@en
P2093
Raymond Lai
Robert J Ingham
P2860
P304
P356
10.1177/2040620712471553
P577
2013-04-01T00:00:00Z