A neuroplasticity hypothesis of chronic stress in the basolateral amygdala.
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Seeding Stress Resilience through InoculationBasolateral amygdala rapid glutamate release encodes an outcome-specific representation vital for reward-predictive cues to selectively invigorate reward-seeking actions.Short environmental enrichment in adulthood reverses anxiety and basolateral amygdala hypertrophy induced by maternal separation.The Role of BDNF in the Development of Fear Learning.Fatty acid amide hydrolase inhibitors produce rapid anti-anxiety responses through amygdala long-term depression in male rodents.Chronic stress, cortisol dysfunction, and pain: a psychoneuroendocrine rationale for stress management in pain rehabilitationNeuroplasticity underlying the comorbidity of pain and depressionThird trimester-equivalent ethanol exposure increases anxiety-like behavior and glutamatergic transmission in the basolateral amygdalaAnalysis of the effects of depression associated polymorphisms on the activity of the BICC1 promoter in amygdala neuronesCircles of engagement: Childhood pain and parent brain.Biophysical modeling of high field diffusion MRI demonstrates micro-structural aberration in chronic mild stress rat brain.
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P2860
A neuroplasticity hypothesis of chronic stress in the basolateral amygdala.
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article científic
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A neuroplasticity hypothesis of chronic stress in the basolateral amygdala.
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A neuroplasticity hypothesis of chronic stress in the basolateral amygdala.
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A neuroplasticity hypothesis of chronic stress in the basolateral amygdala.
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P2860
P921
P1476
A neuroplasticity hypothesis of chronic stress in the basolateral amygdala.
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P2093
Lara M Boyle
P2860
P304
P577
2013-06-01T00:00:00Z
2013-06-13T00:00:00Z