Maladaptive modifications in myofilament proteins and triggers in the progression to heart failure and sudden death. - Wikidata - awgldk - TriplyDB

Maladaptive modifications in myofilament proteins and triggers in the progression to heart failure and sudden death.

Maladaptive modifications in myofilament proteins and triggers in the progression to heart failure and sudden death.

http://www.wikidata.org/entity/Q38184402

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Sarcomere length dependent effects on the interaction between cTnC and cTnI in skinned papillary muscle strips.Liver Kinase B1 complex acts as a novel modifier of myofilament function and localizes to the Z-disk in cardiac myocytes Sex dimorphisms of crossbridge cycling kinetics in transgenic hypertrophic cardiomyopathy mice Dual Labeling Biotin Switch Assay to Reduce Bias Derived From Different Cysteine Subpopulations: A Method to Maximize S-Nitrosylation Detection.Contribution of Post-translational Phosphorylation to Sarcomere-Linked Cardiomyopathy Phenotypes.Commentary: Next Generation Sequencing and Linkage Analysis for the Molecular Diagnosis of a Novel Overlapping Syndrome Characterized by Hypertrophic Cardiomyopathy and Typical Electrical Instability of Brugada Syndrome.Cardiac myofibrillar contractile properties during the progression from hypertension to decompensated heart failure.Calcium in Brugada Syndrome: Questions for Future Research

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Maladaptive modifications in myofilament proteins and triggers in the progression to heart failure and sudden death.

http://www.wikidata.org/entity/Q38184402

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Maladaptive modifications in m ...... eart failure and sudden death.

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Maladaptive modifications in m ...... eart failure and sudden death.

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Pfluegers Archiv

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Maladaptive modifications in m ...... eart failure and sudden death.

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Michelle M Monasky

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R John Solaro

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P2860

Novel control of cardiac myofilament response to calcium by S-glutathionylation at specific sites of myosin binding protein C

Alpha-tropomyosin and cardiac troponin T mutations cause familial hypertrophic cardiomyopathy: a disease of the sarcomere

Uncoupled cardiac nitric oxide synthase mediates diastolic dysfunction

Crystal structure of tropomyosin at 7 Angstroms resolution

Deciphering the design of the tropomyosin molecule

Regulation of the interaction between actin and myosin subfragment 1: evidence for three states of the thin filament

A molecular basis for familial hypertrophic cardiomyopathy: a beta cardiac myosin heavy chain gene missense mutation

Ranolazine improves cardiac diastolic dysfunction through modulation of myofilament calcium sensitivity

Tetrahydrobiopterin improves diastolic dysfunction by reversing changes in myofilament properties

Perturbed length-dependent activation in human hypertrophic cardiomyopathy with missense sarcomeric gene mutations

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1189-1197

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10.1007/S00424-014-1457-7

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