Stochastic genome-nuclear lamina interactions: modulating roles of Lamin A and BAF.
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Anchoring a Leviathan: How the Nuclear Membrane Tethers the GenomeThe second decade of 3C technologies: detailed insights into nuclear organizationPhysiological and Pathological Aging Affects Chromatin Dynamics, Structure and Function at the Nuclear EdgeResetting a functional G1 nucleus after mitosisMAPK signaling pathways and HDAC3 activity are disrupted during differentiation of emerin-null myogenic progenitor cells.Nucleolus association of chromosomal domains is largely maintained in cellular senescence despite massive nuclear reorganisation.Nuclear envelope and genome interactions in cell fate.Barrier-to-Autointegration Factor 1 (BAF/BANF1) Promotes Association of the SETD1A Histone Methyltransferase with Herpes Simplex Virus Immediate-Early Gene Promoters.Global loss of a nuclear lamina component, lamin A/C, and LINC complex components SUN1, SUN2, and nesprin-2 in breast cancer.Perspectives: using polymer modeling to understand the formation and function of nuclear compartments.Dicer interacts with SIRT7 and regulates H3K18 deacetylation in response to DNA damaging agents.Diverse lamin-dependent mechanisms interact to control chromatin dynamics. Focus on laminopathiesStructural organization of human replication timing domains.The redundancy of the mammalian heterochromatic compartment.Lamins in the nuclear interior - life outside the lamina.A-type lamins bind both hetero- and euchromatin, the latter being regulated by lamina-associated polypeptide 2 alpha.Lamin A/C sustains PcG protein architecture, maintaining transcriptional repression at target genes.The nuclear lamina in health and disease.Functional redundancy in the nuclear compartmentalization of the late-replicating genomeThe Molecular Revolution in Cutaneous Biology: Chromosomal Territories, Higher-Order Chromatin Remodeling, and the Control of Gene Expression in Keratinocytes.Lamina-Associated Domains: Links with Chromosome Architecture, Heterochromatin, and Gene Repression.Nuclear lamins are not required for lamina-associated domain organization in mouse embryonic stem cells.Nucleoplasmic Lamin A/C and Polycomb group of proteins: An evolutionarily conserved interplay.Single-cell absolute contact probability detection reveals chromosomes are organized by multiple low-frequency yet specific interactions.Lipodystrophic laminopathy: Lamin A mutation relaxes chromatin architecture to impair adipogenesis.Emerin modulates spatial organization of chromosome territories in cells on softer matrices.Laminopathy-causing lamin A mutations reconfigure lamina-associated domains and local spatial chromatin conformation.SUN2 Modulates HIV-1 Infection and Latency through Association with Lamin A/C To Maintain the Repressive Chromatin.
P2860
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P2860
Stochastic genome-nuclear lamina interactions: modulating roles of Lamin A and BAF.
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
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artículo científico
@es
name
Stochastic genome-nuclear lamina interactions: modulating roles of Lamin A and BAF.
@en
type
label
Stochastic genome-nuclear lamina interactions: modulating roles of Lamin A and BAF.
@en
prefLabel
Stochastic genome-nuclear lamina interactions: modulating roles of Lamin A and BAF.
@en
P2860
P921
P356
P1433
P1476
Stochastic genome-nuclear lamina interactions: modulating roles of Lamin A and BAF.
@en
P2093
P2860
P304
P356
10.4161/NUCL.28825
P577
2014-03-01T00:00:00Z
2014-04-09T00:00:00Z