Myc induced replicative stress response: How to cope with it and exploit it.
about
Trial Watch: Targeting ATM-CHK2 and ATR-CHK1 pathways for anticancer therapyThe Spi1/PU.1 transcription factor accelerates replication fork progression by increasing PP1 phosphatase in leukemiaMolecular Pathways: Overcoming Radiation Resistance by Targeting DNA Damage Response Pathways.Concerted Action of PGC-1-related Coactivator (PRC) and c-MYC in the Stress Response to Mitochondrial Dysfunction.Cancer-Specific Synthetic Lethality between ATR and CHK1 Kinase Activities.PARP inhibitors enhance replication stress and cause mitotic catastrophe in MYCN-dependent neuroblastoma.Perturbations in the Replication Program Contribute to Genomic Instability in Cancer.The MRN complex is transcriptionally regulated by MYCN during neural cell proliferation to control replication stress.The cohesin complex prevents Myc-induced replication stress.A MYCN-MRN complex axis controls replication stress for the safe expansion of neuroprogenitor cells.Around and beyond 53BP1 Nuclear Bodies.Myc inhibits JNK-mediated cell death in vivo.Pan-cancer Alterations of the MYC Oncogene and Its Proximal Network across the Cancer Genome Atlas.
P2860
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P2860
Myc induced replicative stress response: How to cope with it and exploit it.
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article científic
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article scientifique
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articol științific
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articolo scientifico
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artigo científico
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artigo científico
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artigo científico
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artikel ilmiah
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artículo científico
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Myc induced replicative stress response: How to cope with it and exploit it.
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type
label
Myc induced replicative stress response: How to cope with it and exploit it.
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prefLabel
Myc induced replicative stress response: How to cope with it and exploit it.
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P1476
Myc induced replicative stress response: How to cope with it and exploit it.
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P304
P356
10.1016/J.BBAGRM.2014.04.008
P407
P577
2014-04-13T00:00:00Z