Sepsis-induced immunoparalysis: mechanisms, markers, and treatment options.
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Potential survival benefit of polymyxin B hemoperfusion in patients with septic shock: a propensity-matched cohort study.Gamma-irradiated bacille Calmette-Guérin vaccination does not modulate the innate immune response during experimental human endotoxemia in adult malesSoluble B and T Lymphocyte Attenuator Correlates to Disease Severity in Sepsis and High Levels Are Associated with an Increased Risk of MortalityStimulated Whole Blood Cytokine Release as a Biomarker of Immunosuppression in the Critically Ill: The Need for a Standardized Methodology.Intravenous Endotoxin Challenge in Healthy Humans: An Experimental Platform to Investigate and Modulate Systemic Inflammation.Acute Respiratory Distress Syndrome Neutrophils Have a Distinct Phenotype and Are Resistant to Phosphoinositide 3-Kinase Inhibition.Comparison of monocyte human leukocyte antigen-DR expression and stimulated tumor necrosis factor alpha production as outcome predictors in severe sepsis: a prospective observational study.Lactate and Immunosuppression in Sepsis.Human leucocyte antigen (HLA-DR) gene expression is reduced in sepsis and correlates with impaired TNFα response: A diagnostic tool for immunosuppression?Gut Microbial Membership Modulates CD4 T Cell Reconstitution and Function after Sepsis.Hyporesponsiveness of natural killer cells and impaired inflammatory responses in critically ill patients.The Effects of Ex Vivo Administration of Granulocyte-Macrophage Colony-Stimulating Factor and Endotoxin on Cytokine Release of Whole Blood Are Determined by Priming Conditions.Short-term repeated HRV-16 exposure results in an attenuated immune response in vivo in humans.Dynamics of monocytic HLA-DR expression differs between bacterial etiologies during the course of bloodstream infection.Monocyte Function and Clinical Outcomes in Febrile and Afebrile Patients with Severe Sepsis.Pro-Inflammatory Th1 and Th17 Cells Are Suppressed During Human Experimental Endotoxemia Whereas Anti-Inflammatory IL-10 Producing T-Cells Are Unaffected.Stroma: the forgotten cells of innate immune memory.Precision Immunotherapy for SepsisThe CTLA-4 rs231775 GG genotype is associated with favorable 90-day survival in Caucasian patients with sepsisMicroRNAs 143 and 150 in whole blood enable detection of T-cell immunoparalysis in sepsisHypothesis: Fever control, a niche for alpha-2 agonists in the setting of septic shock and severe acute respiratory distress syndrome?
P2860
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P2860
Sepsis-induced immunoparalysis: mechanisms, markers, and treatment options.
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article científic
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article scientifique
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articol științific
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articolo scientifico
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artigo científico
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artigo científico
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Sepsis-induced immunoparalysis: mechanisms, markers, and treatment options.
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label
Sepsis-induced immunoparalysis: mechanisms, markers, and treatment options.
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prefLabel
Sepsis-induced immunoparalysis: mechanisms, markers, and treatment options.
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P2093
P1476
Sepsis-induced immunoparalysis: mechanisms, markers, and treatment options.
@en
P2093
P304
P577
2014-05-30T00:00:00Z