SOS, the formidable strategy of bacteria against aggressions.
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SOS response and its regulation on the fluoroquinolone resistance.Review of Antimicrobial Resistance in the Environment and Its Relevance to Environmental RegulatorsComparative Genomics of DNA Recombination and Repair in Cyanobacteria: Biotechnological ImplicationsPlasmid-mediated quinolone resistance in Enterobacteriaceae: a systematic review with a focus on Mediterranean countries.The regulatory function of LexA is temperature-dependent in the deep-sea bacterium Shewanella piezotolerans WP3Antibiotics trigger initiation of SCCmec transfer by inducing SOS responses.Exploiting CRISPR-Cas to manipulate Enterococcus faecalis populations.Lack of AcrB Efflux Function Confers Loss of Virulence on Salmonella enterica Serovar Typhimurium.Label-free proteomic analysis to confirm the predicted proteome of Corynebacterium pseudotuberculosis under nitrosative stress mediated by nitric oxide.Stationary-Phase Persisters to Ofloxacin Sustain DNA Damage and Require Repair Systems Only during Recovery.Antimicrobial Susceptibility and SOS-Dependent Increase in Mutation Frequency Are Impacted by Escherichia coli Topoisomerase I C-Terminal Point Mutation.The Bacterial Mfd Protein Prevents DNA Damage Induced by the Host Nitrogen Immune Response in a NER-Independent but RecBC-Dependent PathwayAnalysis of LexA binding sites and transcriptomics in response to genotoxic stress in Leptospira interrogans.Antibiotic stress selects against cooperation in the pathogenic bacterium Pseudomonas aeruginosa.Antibiotic Capture by Bacterial Lipocalins Uncovers an Extracellular Mechanism of Intrinsic Antibiotic ResistanceBeyond xeroderma pigmentosum: DNA damage and repair in an ecological context. A tribute to James E. Cleaver.Effect of LexA on Chromosomal Integration of CTXϕ in Vibrio cholerae.How much territory can a single E. coli cell control?Increased ParB level affects expression of stress response, adaptation and virulence operons and potentiates repression of promoters adjacent to the high affinity binding sites parS3 and parS4 in Pseudomonas aeruginosa.Efflux pump induction by quaternary ammonium compounds and fluoroquinolone resistance in bacteria.Radiation-induced translational control of gene expression.Targeting bacterial topoisomerases: how to counter mechanisms of resistance.Mechanisms of bacterial persistence during stress and antibiotic exposure.Intracellular d-Serine Accumulation Promotes Genetic Diversity via Modulated Induction of RecA in Enterohemorrhagic Escherichia coli.The risk of low concentrations of antibiotics in agriculture for resistance in human health care.Genomic survey and expression analysis of DNA repair genes in the genus Leptospira.Differential roles of antimicrobials in the acquisition of drug resistance through activation of the SOS response in Acinetobacter baumannii.Cellular Response to Ciprofloxacin in Low-Level Quinolone-Resistant Escherichia coli.NQO-Induced DNA-Less Cell Formation Is Associated with Chromatin Protein Degradation and Dependent on A0A1-ATPase in Sulfolobus.Transfer and Persistence of a Multi-Drug Resistance Plasmid in situ of the Infant Gut Microbiota in the Absence of Antibiotic TreatmentPotential impacts of aquatic pollutants: sub-clinical antibiotic concentrations induce genome changes and promote antibiotic resistance.Quinolone Resistance Reversion by Targeting the SOS Response.Subinhibitory Concentrations of Ciprofloxacin Enhance Antimicrobial Resistance and Pathogenicity of Enterococcus faecium.A loopy view of telomere evolution.A programmed cell division delay preserves genome integrity during natural genetic transformation in Streptococcus pneumoniae.DNA damage responses and stress resistance: Concepts from bacterial SOS to metazoan immunity.Prophages and Growth Dynamics Confound Experimental Results with Antibiotic-Tolerant Persister Cells.Risk for the development of Antimicrobial Resistance (AMR) due to feeding of calves with milk containing residues of antibioticsSOS gene induction and possible mutagenic effects of freeze-drying in Escherichia coli and Salmonella typhimurium.Staphylococcus aureus requires at least one FtsK/SpoIIIE protein for correct chromosome segregation.
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SOS, the formidable strategy of bacteria against aggressions.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年学术文章
@wuu
2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
@yue
2014年學術文章
@zh
2014年學術文章
@zh-hant
name
SOS, the formidable strategy of bacteria against aggressions.
@en
type
label
SOS, the formidable strategy of bacteria against aggressions.
@en
prefLabel
SOS, the formidable strategy of bacteria against aggressions.
@en
P2860
P356
P1476
SOS, the formidable strategy of bacteria against aggressions
@en
P2093
Didier Mazel
Zeynep Baharoglu
P2860
P304
P356
10.1111/1574-6976.12077
P577
2014-06-30T00:00:00Z