How to make a heart valve: from embryonic development to bioengineering of living valve substitutes. - Wikidata - awgldk - TriplyDB

How to make a heart valve: from embryonic development to bioengineering of living valve substitutes.

How to make a heart valve: from embryonic development to bioengineering of living valve substitutes.

http://www.wikidata.org/entity/Q38264871

about

klf2a couples mechanotransduction and zebrafish valve morphogenesis through fibronectin synthesis.Wnt/β-catenin signaling enables developmental transitions during valvulogenesis.Current progress in tissue engineering of heart valves: multiscale problems, multiscale solutions Effect of altered haemodynamics on the developing mitral valve in chick embryonic heart.Morphology, Clinicopathologic Correlations, and Mechanisms in Heart Valve Health and Disease.HAND2 Target Gene Regulatory Networks Control Atrioventricular Canal and Cardiac Valve Development.Hemodynamic Forces Sculpt Developing Heart Valves through a KLF2-WNT9B Paracrine Signaling Axis.Tissue-Engineered Tubular Heart Valves Combining a Novel Precontraction Phase with the Self-Assembly Method.Biomaterial-driven in situ cardiovascular tissue engineering-a multi-disciplinary perspective.Monocytes and Macrophages in Heart Valves: Uninvited Guests or Critical Performers?Sheep-Specific Immunohistochemical Panel for the Evaluation of Regenerative and Inflammatory Processes in Tissue-Engineered Heart Valves

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How to make a heart valve: from embryonic development to bioengineering of living valve substitutes.

http://www.wikidata.org/entity/Q38264871

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2014 nî lūn-bûn

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2014年の論文

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2014年学术文章

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2014年学术文章

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2014年學術文章

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2014年學術文章

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2014年學術文章

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Cold Spring Harbor Perspectives in Medicine

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How to make a heart valve: fro ...... g of living valve substitutes.

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Anita Driessen-Mol

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Frank Baaijens

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Hesr1 and Hesr2 regulate atrioventricular boundary formation in the developing heart through the repression of Tbx2

Twist1 promotes heart valve cell proliferation and extracellular matrix gene expression during development in vivo and is expressed in human diseased aortic valves

Integration of a Notch-dependent mesenchymal gene program and Bmp2-driven cell invasiveness regulates murine cardiac valve formation.

Mutations in NOTCH1 cause aortic valve disease

Slug is a direct Notch target required for initiation of cardiac cushion cellularization

Coordinating tissue interactions: Notch signaling in cardiac development and disease

Endocardial and epicardial epithelial to mesenchymal transitions in heart development and disease

Epithelial-mesenchymal transitions: insights from development

Stem cells for heart valve regeneration

Development of heart valves requires Gata4 expression in endothelial-derived cells

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11

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José Luis De La Pompa Mínguez

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Guillermo Luxán

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2014-11-03T00:00:00Z

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