SnoN as a novel negative regulator of TGF-β/Smad signaling: a target for tailoring organ fibrosis.
about
TGF-β receptor 1 inhibition prevents stenosis of tissue-engineered vascular grafts by reducing host mononuclear phagocyte activation.TGFβ1 regulates Scleraxis expression in primary cardiac myofibroblasts by a Smad-independent mechanism.Activation of SIRT3 by resveratrol ameliorates cardiac fibrosis and improves cardiac function via the TGF-β/Smad3 pathway.Herbal compound 861 prevents hepatic fibrosis by inhibiting the TGF-β1/Smad/SnoN pathway in bile duct-ligated rats.Down-regulation of miR-23a inhibits high glucose-induced EMT and renal fibrogenesis by up-regulation of SnoN.Matrine suppresses cardiac fibrosis by inhibiting the TGF‑β/Smad pathway in experimental diabetic cardiomyopathy.
P2860
SnoN as a novel negative regulator of TGF-β/Smad signaling: a target for tailoring organ fibrosis.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年学术文章
@wuu
2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
@yue
2014年學術文章
@zh
2014年學術文章
@zh-hant
name
SnoN as a novel negative regul ...... for tailoring organ fibrosis.
@en
type
label
SnoN as a novel negative regul ...... for tailoring organ fibrosis.
@en
prefLabel
SnoN as a novel negative regul ...... for tailoring organ fibrosis.
@en
P2860
P50
P1476
SnoN as a novel negative regul ...... t for tailoring organ fibrosis
@en
P2093
Mark Hnatowich
P2860
P304
P356
10.1152/AJPHEART.00453.2014
P577
2014-11-07T00:00:00Z