Delayed parturition and altered myometrial progesterone receptor isoform A expression in mice null for Krüppel-like factor 9.
about
The Krüppel-like factor 9 (KLF9) network in HEC-1-A endometrial carcinoma cells suggests the carcinogenic potential of dys-regulated KLF9 expressionThe Krüppel-like factors in female reproductive system pathologiesNrk, an X-linked protein kinase in the germinal center kinase family, is required for placental development and fetoplacental induction of laborNrf2 amplifies oxidative stress via induction of Klf9.An evolutionary test of the isoform switching hypothesis of functional progesterone withdrawal for parturition: humans have a weaker repressive effect of PR-A than mice.Response of adult mouse uterus to early disruption of estrogen receptor-alpha signaling is influenced by Krüppel-like factor 9Prolonged pregnancy in women is associated with attenuated myometrial expression of progesterone receptor co-regulator Krüppel-like Factor 9.Genomics of preterm birth.Krüppel-like factor 9 loss-of-expression in human endometrial carcinoma links altered expression of growth-regulatory genes with aberrant proliferative response to estrogen.The reproductive phenotype of mice null for transcription factor Krüppel-like factor 13 suggests compensatory function of family member Krüppel-like factor 9 in the peri-implantation uterusThe T3-induced gene KLF9 regulates oligodendrocyte differentiation and myelin regeneration.KLF9 is a novel transcriptional regulator of bortezomib- and LBH589-induced apoptosis in multiple myeloma cells.Extensive shift in placental transcriptome profile in preeclampsia and placental origin of adverse pregnancy outcomesParturition failure in mice lacking Mamld1.Krüppel-Like Factor 13 Deficiency in Uterine Endometrial Cells Contributes to Defective Steroid Hormone Receptor Signaling but Not Lesion Establishment in a Mouse Model of EndometriosisInsights into parturition biology from genetically altered mice.Minireview: fetal-maternal hormonal signaling in pregnancy and labor.Krüppel-like factor 9 deficiency in uterine endometrial cells promotes ectopic lesion establishment associated with activated notch and hedgehog signaling in a mouse model of endometriosis.Progesterone Receptor Signaling in Uterine Myometrial Physiology and Preterm Birth.In vivo Raman spectral analysis of impaired cervical remodeling in a mouse model of delayed parturitionProstaglandin-Endoperoxide Synthase 1 Mediates the Timing of Parturition in Mice Despite Unhindered Uterine Contractility.
P2860
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P2860
Delayed parturition and altered myometrial progesterone receptor isoform A expression in mice null for Krüppel-like factor 9.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
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2008年學術文章
@zh-hant
name
Delayed parturition and altere ...... ull for Krüppel-like factor 9.
@en
type
label
Delayed parturition and altere ...... ull for Krüppel-like factor 9.
@en
prefLabel
Delayed parturition and altere ...... ull for Krüppel-like factor 9.
@en
P2093
P2860
P1476
Delayed parturition and altere ...... ull for Krüppel-like factor 9.
@en
P2093
Frank A Simmen
Michael C Velarde
Rosalia C M Simmen
Zhaoyang Zeng
P2860
P304
P356
10.1095/BIOLREPROD.107.065821
P407
P577
2008-02-27T00:00:00Z