Repression of inflammatory responses in the absence of DNA binding by the glucocorticoid receptor. - Wikidata - awgldk - TriplyDB

Repression of inflammatory responses in the absence of DNA binding by the glucocorticoid receptor.

Repression of inflammatory responses in the absence of DNA binding by the glucocorticoid receptor.

http://www.wikidata.org/entity/Q38293939

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Regulation of IkappaBalpha function and NF-kappaB signaling: AEBP1 is a novel proinflammatory mediator in macrophages Dexamethasone causes sustained expression of mitogen-activated protein kinase (MAPK) phosphatase 1 and phosphatase-mediated inhibition of MAPK p38.A nuclear isoform of the focal adhesion LIM-domain protein Trip6 integrates activating and repressing signals at AP-1- and NF-kappaB-regulated promoters Dissociation of transactivation from transrepression by a selective glucocorticoid receptor agonist leads to separation of therapeutic effects from side effects The use of inhaled corticosteroids in pediatric asthma: update Specificity and sensitivity of glucocorticoid signaling in health and disease Glucocorticoid regulation of inflammation and its functional correlates: from HPA axis to glucocorticoid receptor dysfunction Glucocorticoids in the management of peritumoral brain edema: a review of molecular mechanisms Compound A, a dissociated glucocorticoid receptor modulator, inhibits T-bet (Th1) and induces GATA-3 (Th2) activity in immune cells VBP15, a glucocorticoid analogue, is effective at reducing allergic lung inflammation in mice A CD28 superagonistic antibody elicits 2 functionally distinct waves of T cell activation in rats.The orphan nuclear receptor SHP acts as a negative regulator in inflammatory signaling triggered by Toll-like receptors Evaluation of the selective glucocorticoid receptor agonist compound A for ototoxic effects Hypothalamic-pituitary-adrenal axis dysregulation and behavioral analysis of mouse mutants with altered glucocorticoid or mineralocorticoid receptor function Therapeutic and adverse effects of a non-steroidal glucocorticoid receptor ligand in a mouse model of multiple sclerosis.Prednisolone-induced differential gene expression in mouse liver carrying wild type or a dimerization-defective glucocorticoid receptor.The human glucocorticoid receptor: molecular basis of biologic function Crosstalk in inflammation: the interplay of glucocorticoid receptor-based mechanisms and kinases and phosphatases Transrepression function of the glucocorticoid receptor regulates eyelid development and keratinocyte proliferation but is not sufficient to prevent skin chronic inflammation.Minireview: latest perspectives on antiinflammatory actions of glucocorticoids.Abrogation of glucocorticoid receptor dimerization correlates with dissociated glucocorticoid behavior of compound a.Target-specific utilization of transcriptional regulatory surfaces by the glucocorticoid receptor.A role for endogenous glucocorticoids in wound repair Transcription factor AP1 potentiates chromatin accessibility and glucocorticoid receptor binding Control of macrophage activation and function by PPARs.Inhibition of nuclear factor-kappaB ameliorates bowel injury and prolongs survival in a neonatal rat model of necrotizing enterocolitis.TNFα and IFNγ synergistically enhance transcriptional activation of CXCL10 in human airway smooth muscle cells via STAT-1, NF-κB, and the transcriptional coactivator CREB-binding protein.Increased p300 expression inhibits glucocorticoid receptor-T-cell receptor antagonism but does not affect thymocyte positive selection Interferon-inducible factor 16 is a novel modulator of glucocorticoid action Going nuclear in metabolic and cardiovascular disease Glucocorticoid-targeted therapies for the treatment of rheumatoid arthritis.Minireview: rapid glucocorticoid signaling via membrane-associated receptors Impact of glucocorticoid receptor density on ligand-independent dimerization, cooperative ligand-binding and basal priming of transactivation: a cell culture model.Scientific and clinical challenges in sepsis.Glucocorticoid-independent repression of tumor necrosis factor (TNF) alpha-stimulated interleukin (IL)-6 expression by the glucocorticoid receptor: a potential mechanism for protection against an excessive inflammatory response.The regulation of muscle mass by endogenous glucocorticoids.Role of metabolically active hormones in the insulin resistance associated with short-term glucocorticoid treatment.Glucocorticoid receptor alpha isoform-selective regulation of antiapoptotic genes in osteosarcoma cells: a new mechanism for glucocorticoid resistance.Nontranscriptional actions of the glucocorticoid receptor.Circadian CLOCK-mediated regulation of target-tissue sensitivity to glucocorticoids: implications for cardiometabolic diseases

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Repression of inflammatory responses in the absence of DNA binding by the glucocorticoid receptor.

http://www.wikidata.org/entity/Q38293939

description

2001 nî lūn-bûn

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2001年の論文

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2001年学术文章

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Repression of inflammatory res ...... y the glucocorticoid receptor.

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Repression of inflammatory res ...... y the glucocorticoid receptor.

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Repression of inflammatory res ...... y the glucocorticoid receptor.

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Repression of inflammatory res ...... y the glucocorticoid receptor.

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Angel P

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Göttlicher M

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Herrlich P

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Tuckermann JP

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Vujic M

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Weih F

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P2860

The glucocorticoid receptor inhibits NFkappaB by interfering with serine-2 phosphorylation of the RNA polymerase II carboxy-terminal domain

The steroid and thyroid hormone receptor superfamily

Functional interactions between Stat5 and the glucocorticoid receptor

Negative cross-talk between RelA and the glucocorticoid receptor: a possible mechanism for the antiinflammatory action of glucocorticoids

Steroid hormone receptors: many actors in search of a plot

NF-kappa B: ten years after

Cross-talk between glucocorticoid receptor and AP-1.

DNA binding of the glucocorticoid receptor is not essential for survival.

The DNA binding-independent function of the glucocorticoid receptor mediates repression of AP-1-dependent genes in skin.

Glucocorticoid-induced apoptosis of human leukemic cells is caused by the repressive function of the glucocorticoid receptor

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