A central role for neuronal AMP-activated protein kinase (AMPK) and mammalian target of rapamycin (mTOR) in high-protein diet-induced weight loss.
about
AMP-activated protein kinase, stress responses and cardiovascular diseasesHypothalamic AMPK as a Regulator of Energy HomeostasisReviewing the Effects of L-Leucine Supplementation in the Regulation of Food Intake, Energy Balance, and Glucose HomeostasisDietary leucine--an environmental modifier of insulin resistance acting on multiple levels of metabolismCentral Amino Acid Sensing in the Control of Feeding BehaviorProtein-dependent regulation of feeding and metabolismThe action of leptin in the ventral tegmental area to decrease food intake is dependent on Jak-2 signalingThe role of hypothalamic mammalian target of rapamycin complex 1 signaling in diet-induced obesity.Central exercise action increases the AMPK and mTOR response to leptinMacronutrient balance and lifespanLeucine acts in the brain to suppress food intake but does not function as a physiological signal of low dietary protein.DEP domain-containing mTOR-interacting protein in the rat brain: distribution of expression and potential implication.Hypothalamic mTOR signaling mediates the orexigenic action of ghrelin.Higher branched-chain amino acid intake is associated with a lower prevalence of being overweight or obese in middle-aged East Asian and Western adults.AMPK as Target for Intervention in Childhood and Adolescent ObesityB(0)AT2 (SLC6A15) is localized to neurons and astrocytes, and is involved in mediating the effect of leucine in the brainLeucine supplementation protects from insulin resistance by regulating adiposity levelsFish oil promotes survival and protects against cognitive decline in severely undernourished mice by normalizing satiety signals.Oral leucine supplementation is sensed by the brain but neither reduces food intake nor induces an anorectic pattern of gene expression in the hypothalamus.Pregnancy induces resistance to the anorectic effect of hypothalamic malonyl-CoA and the thermogenic effect of hypothalamic AMPK inhibition in female rats.AMPK inhibition in health and disease.Leucine supplementation improves adiponectin and total cholesterol concentrations despite the lack of changes in adiposity or glucose homeostasis in rats previously exposed to a high-fat diet.Homeostatic regulation of protein intake: in search of a mechanismImpaired branched chain amino acid metabolism alters feeding behavior and increases orexigenic neuropeptide expression in the hypothalamusLinking Gut Microbiota and Inflammation to Obesity and Insulin Resistance.Dietary whey protein lessens several risk factors for metabolic diseases: a review.Leucine supplementation increases SIRT1 expression and prevents mitochondrial dysfunction and metabolic disorders in high-fat diet-induced obese mice.Brainstem nutrient sensing in the nucleus of the solitary tract inhibits feeding.Neural and metabolic regulation of macronutrient intake and selection.Brain responses to high-protein dietsEffects of metformin and sitagliptin on glycolipid metabolism in type 2 diabetic rats on different dietsHepatic autophagy contributes to the metabolic response to dietary protein restrictionAnorexigenic and orexigenic hormone modulation of mammalian target of rapamycin complex 1 activity and the regulation of hypothalamic agouti-related protein mRNA expression.Adipose-immune interactions during obesity and caloric restriction: reciprocal mechanisms regulating immunity and health spanThe Macronutrients, Appetite, and Energy Intake.Prunus mume and Lithospermum erythrorhizon Extracts Synergistically Prevent Visceral Adiposity by Improving Energy Metabolism through Potentiating Hypothalamic Leptin and Insulin Signalling in Ovariectomized Rats.Evidence for central regulation of glucose metabolism.Structure and physiological actions of ghrelinCNS regulation of glucose homeostasis.Beyond the role of dietary protein and amino acids in the prevention of diet-induced obesity.
P2860
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P2860
A central role for neuronal AMP-activated protein kinase (AMPK) and mammalian target of rapamycin (mTOR) in high-protein diet-induced weight loss.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
A central role for neuronal AM ...... tein diet-induced weight loss.
@en
type
label
A central role for neuronal AM ...... tein diet-induced weight loss.
@en
prefLabel
A central role for neuronal AM ...... tein diet-induced weight loss.
@en
P2093
P50
P356
P1433
P1476
A central role for neuronal AM ...... otein diet-induced weight loss
@en
P2093
Eduardo R Ropelle
Joseane Morari
José B C Carvalheira
José R Pauli
Kellen K Souza
Kleber G Franchini
Lício A Velloso
Maria Fernanda A Fernandes
Mario J A Saad
Marília M Dias
P304
P356
10.2337/DB07-0573
P407
P577
2007-12-05T00:00:00Z