Diabetic KKAy mice exhibit increased hepatic PPARgamma1 gene expression and develop hepatic steatosis upon chronic treatment with antidiabetic thiazolidinediones.
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Gene expression profiling of potential peroxisome proliferator-activated receptor (PPAR) target genes in human hepatoblastoma cell lines inducibly expressing different PPAR isoformsEvidence-based selection of training compounds for use in the mechanism-based integrated prediction of drug-induced liver injury in manGlial cell line-derived neurotrophic factor protects against high-fat diet-induced hepatic steatosis by suppressing hepatic PPAR-γ expressionHas natural selection in human populations produced two types of metabolic syndrome (with and without fatty liver)?Peroxisome proliferator activated receptor-gamma in pathogenesis of experimental fatty liver disease.Anti-obesity activity of diglyceride containing conjugated linoleic acid in C57BL/6J ob/ob mice.HVC1 ameliorates hyperlipidemia and inflammation in LDLR-/- mice.Early hepatic insulin resistance precedes the onset of diabetes in obese C57BLKS-db/db mice.Gene Expression Changes Induced by PPAR Gamma Agonists in Animal and Human Liver.PPAR gamma and human metabolic diseaseGhrelin promotes hepatic lipogenesis by activation of mTOR-PPARγ signaling pathway.Protopanaxatriol, a novel PPARγ antagonist from Panax ginseng, alleviates steatosis in mice.Liver-specific disruption of PPARgamma in leptin-deficient mice improves fatty liver but aggravates diabetic phenotypes.Long-term ethanol consumption promotes hepatic tumorigenesis but impairs normal hepatocyte proliferation in rats.LPSF/GQ-02 inhibits the development of hepatic steatosis and inflammation in a mouse model of non-alcoholic fatty liver disease (NAFLD).Upregulation of Scavenger Receptor BI by Hepatic Nuclear Factor 4α through a Peroxisome Proliferator-Activated Receptor γ-Dependent Mechanism in Liver.Genome-wide profiling of liver X receptor, retinoid X receptor, and peroxisome proliferator-activated receptor α in mouse liver reveals extensive sharing of binding sitesBone is a target for the antidiabetic compound rosiglitazone.Effect of Chronic Pioglitazone Treatment on Hepatic Gene Expression Profile in Obese C57BL/6J Mice.Pleiotropic effects of thiazolidinediones: taking a look beyond antidiabetic activity.Diet high in fat and sucrose induces rapid onset of obesity-related metabolic syndrome partly through rapid response of genes involved in lipogenesis, insulin signalling and inflammation in mice.Modulation Effect of Peroxisome Proliferator-Activated Receptor Agonists on Lipid Droplet Proteins in Liver.WAT is a functional adipocyte?Hepatic steatosis in leptin-deficient mice is promoted by the PPARgamma target gene Fsp27.Hexarelin Signaling to PPARgamma in Metabolic Diseases.ATF4 protein deficiency protects against high fructose-induced hypertriglyceridemia in mice.Reduced energy expenditure and increased inflammation are early events in the development of ovariectomy-induced obesity.P633H, a novel dual agonist at peroxisome proliferator-activated receptors alpha and gamma, with different anti-diabetic effects in db/db and KK-Ay mice.Herpesvirus-associated ubiquitin-specific protease (HAUSP) modulates peroxisome proliferator-activated receptor γ (PPARγ) stability through its deubiquitinating activity.PPAR and liver injury in HIV-infected patients.FoxO6 integrates insulin signaling with MTP for regulating VLDL production in the liver.Mechanisms by which the thiazolidinedione troglitazone protects against sucrose-induced hepatic fat accumulation and hyperinsulinaemiaEffects and Potential Mechanisms of Pioglitazone on Lipid Metabolism in Obese Diabetic KKAy Mice.Which CIDE are you on? Apoptosis and energy metabolism.Peroxisome proliferator-activated receptor-γ as a therapeutic target for hepatic fibrosis: from bench to bedside.Fish oil prevents excessive accumulation of subcutaneous fat caused by an adverse effect of pioglitazone treatment and positively changes adipocytes in KK mice.Update on pparγ and nonalcoholic Fatty liver disease.Ω-3 fatty acids prevent hepatic steatosis, independent of PPAR-α activity, in a murine model of parenteral nutrition-associated liver disease.Analysis of the role of hepatic PPARγ expression during mouse liver regeneration.Effect of 2,4-thiazolidinedione on limousin cattle growth and on muscle and adipose tissue metabolism.
P2860
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P2860
Diabetic KKAy mice exhibit increased hepatic PPARgamma1 gene expression and develop hepatic steatosis upon chronic treatment with antidiabetic thiazolidinediones.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
Diabetic KKAy mice exhibit inc ...... tidiabetic thiazolidinediones.
@en
type
label
Diabetic KKAy mice exhibit inc ...... tidiabetic thiazolidinediones.
@en
prefLabel
Diabetic KKAy mice exhibit inc ...... tidiabetic thiazolidinediones.
@en
P2093
P1476
Diabetic KKAy mice exhibit inc ...... tidiabetic thiazolidinediones.
@en
P2093
Atzpodien E
Bedoucha M
Boelsterli UA
P356
10.1016/S0168-8278(01)00066-6
P577
2001-07-01T00:00:00Z