Splice-shifting oligonucleotide (SSO) mediated blocking of an exonic splicing enhancer (ESE) created by the prevalent c.903+469T>C MTRR mutation corrects splicing and restores enzyme activity in patient cells.
about
Oligonucleotide Therapies: The Past and the PresentRole of an SNP in Alternative Splicing of Bovine NCF4 and Mastitis SusceptibilityTargeted Next Generation Sequencing in Patients with Inborn Errors of MetabolismGlobal identification of hnRNP A1 binding sites for SSO-based splicing modulation.Functional SNPs of INCENP Affect Semen Quality by Alternative Splicing Mode and Binding Affinity with the Target Bta-miR-378 in Chinese Holstein Bulls.
P2860
Splice-shifting oligonucleotide (SSO) mediated blocking of an exonic splicing enhancer (ESE) created by the prevalent c.903+469T>C MTRR mutation corrects splicing and restores enzyme activity in patient cells.
description
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name
Splice-shifting oligonucleotid ...... yme activity in patient cells.
@en
type
label
Splice-shifting oligonucleotid ...... yme activity in patient cells.
@en
prefLabel
Splice-shifting oligonucleotid ...... yme activity in patient cells.
@en
P2093
P2860
P50
P356
P1476
Splice-shifting oligonucleotid ...... zyme activity in patient cells
@en
P2093
Brian Fowler
Bruno Palhais
Gitte Hoffmann Bruun
Henriette Skovgaard Andersen
Patricie Burda
Seraina Lutz
Terttu Suormala
Veronica S Præstegaard
P2860
P304
P356
10.1093/NAR/GKV275
P407
P577
2015-04-15T00:00:00Z