Platelets are an unindicted culprit in the development of endometriosis: clinical and experimental evidence.
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Dating Endometriotic Ovarian Cysts Based on the Content of Cyst Fluid and its Potential Clinical ImplicationsAnti-platelet therapy holds promises in treating adenomyosis: experimental evidence.1-Methyl-tryptophan attenuates regulatory T cells differentiation due to the inhibition of estrogen-IDO1-MRC2 axis in endometriosisEnhancer of Zeste homolog 2 (EZH2) induces epithelial-mesenchymal transition in endometriosis.Corroborating evidence for platelet-induced epithelial-mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation in the development of adenomyosis.Origins and Progression of Adolescent Endometriosis.Pharmacologic treatment of the ovarian endometrioma.Platelets impair natural killer cell reactivity and function in endometriosis through multiple mechanisms.Total circulating microparticle levels are increased in patients with deep infiltrating endometriosis.Chronic stress accelerates the development of endometriosis in mouse through adrenergic receptor β2.Cellular Changes Consistent With Epithelial-Mesenchymal Transition and Fibroblast-to-Myofibroblast Transdifferentiation in the Progression of Experimental Endometriosis in Baboons.Transforming growth factor β1 signaling coincides with epithelial-mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation in the development of adenomyosis in mice.Evidence for a Hypercoagulable State in Women With Ovarian Endometriomas.Transvaginal Elastosonography as an Imaging Technique for Diagnosing Adenomyosis.Histological and Immunohistochemical Characterization of the Similarity and Difference Between Ovarian Endometriomas and Deep Infiltrating Endometriosis.Endometriosis-Derived Stromal Cells Secrete Thrombin and Thromboxane A2, Inducing Platelet Activation.Platelet-derived TGF-β1 mediates the down-modulation of NKG2D expression and may be responsible for impaired natural killer (NK) cytotoxicity in women with endometriosis.Cancer driver mutations in endometriosis: Variations on the major theme of fibrogenesis
P2860
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P2860
Platelets are an unindicted culprit in the development of endometriosis: clinical and experimental evidence.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
@yue
2015年論文
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2015年論文
@zh-hk
2015年論文
@zh-mo
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name
Platelets are an unindicted cu ...... cal and experimental evidence.
@en
type
label
Platelets are an unindicted cu ...... cal and experimental evidence.
@en
prefLabel
Platelets are an unindicted cu ...... cal and experimental evidence.
@en
P2093
P2860
P356
P1433
P1476
Platelets are an unindicted cu ...... cal and experimental evidence.
@en
P2093
P2860
P304
P356
10.1093/HUMREP/DEV025
P407
P50
P577
2015-03-03T00:00:00Z