Candida albicans versus Candida dubliniensis: Why Is C. albicans More Pathogenic?
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Telomeric ORFS in Candida albicans: does mediator tail wag the yeast?Purpurin triggers caspase-independent apoptosis in Candida dubliniensis biofilmsAmplification of TLO Mediator Subunit Genes Facilitate Filamentous Growth in Candida SppAntifungal application of nonantifungal drugsEpidemiology, antifungal susceptibility, and pathogenicity of Candida africana isolates from the United Kingdom.Meningitis Caused by Candida Dubliniensis in a Patient with Cirrhosis: A Case Report and Review of the Literature.Candida dubliniensis: an appraisal of its clinical significance as a bloodstream pathogen.Telomeric ORFs (TLOs) in Candida spp. Encode mediator subunits that regulate distinct virulence traits.Fungal mediator tail subunits contain classical transcriptional activation domains.First-time isolation of Candida dubliniensis from plaque and carious dentine of primary teeth.Csr1/Zap1 Maintains Zinc Homeostasis and Influences Virulence in Candida dubliniensis but Is Not Coupled to Morphogenesis.The Tlo proteins are stoichiometric components of Candida albicans mediator anchored via the Med3 subunit.Comparison of Switching and Biofilm Formation between MTL-Homozygous Strains of Candida albicans and Candida dubliniensisPopulation structure and molecular genetic characterization of 5-flucytosine-susceptible and -resistant clinical Candida dubliniensis isolates from Kuwait.Methods of Candida dubliniensis identification and its occurrence in human clinical material.Candida albicans pathogenicity mechanisms.Comparative evolution of morphological regulatory functions in Candida species.Candida dubliniensis endophthalmitis: five cases over 15 years.Molecular Fingerprinting Studies Do Not Support Intrahospital Transmission of Candida albicans among Candidemia Patients in Kuwait.Thriving within the host: Candida spp. interactions with phagocytic cells.Third case of Candida dubliniensis endogenous endophthalmitis in North America: case report and review of the literature.Pathogenicity mechanisms and host response during oral Candida albicans infections.Usefulness of the Non-conventional Caenorhabditis elegans Model to Assess Candida Virulence.Enterococcus faecalis bacteriocin EntV inhibits hyphal morphogenesis, biofilm formation, and virulence of Candida albicans.Enterohemorrhagic Escherichia coli promotes the invasion and tissue damage of enterocytes infected with Candida albicans in vitro.Candida species Rewired Hyphae Developmental Programs for Chlamydospore Formation.The effect of psychoactive substances (drugs) on the presence and frequency of oral Candida species and Candida dubliniensisImpact of photocatalysis on fungal cells: depiction of cellular and molecular effects on Saccharomyces cerevisiae.Down-regulation of the ALS3 gene as a consequent effect of RNA-mediated silencing of the EFG1 gene in Candida albicans.Comparative adherence of Candida albicans and Candida dubliniensis to human buccal epithelial cells and extracellular matrix proteins.Gene flow contributes to diversification of the major fungal pathogen Candida albicans.
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P2860
Candida albicans versus Candida dubliniensis: Why Is C. albicans More Pathogenic?
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Candida albicans versus Candida dubliniensis: Why Is C. albicans More Pathogenic?
@en
type
label
Candida albicans versus Candida dubliniensis: Why Is C. albicans More Pathogenic?
@en
prefLabel
Candida albicans versus Candida dubliniensis: Why Is C. albicans More Pathogenic?
@en
P2860
P50
P356
P1476
Candida albicans versus Candida dubliniensis: Why Is C. albicans More Pathogenic?
@en
P2860
P304
P356
10.1155/2012/205921
P577
2011-09-04T00:00:00Z