Construction and analysis of chromosomal Clostridium difficile mutants.
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Clostridium difficile phages: still difficult?Toxin B is essential for virulence of Clostridium difficileImportance of Glutamate Dehydrogenase (GDH) in Clostridium difficile Colonization In VivoStructural and functional analysis of the CspB protease required for Clostridium spore germinationThe potential for emerging therapeutic options for Clostridium difficile infectionSpoIVA and SipL are Clostridium difficile spore morphogenetic proteinsIntegration of metabolism and virulence by Clostridium difficile CodYRepression of Clostridium difficile toxin gene expression by CodY.Global transcriptional control by glucose and carbon regulator CcpA in Clostridium difficile.Novel system for efficient isolation of Clostridium double-crossover allelic exchange mutants enabling markerless chromosomal gene deletions and DNA integration.Functional analysis of the VirSR phosphorelay from Clostridium perfringens.Truncation in the tcdC region of the Clostridium difficile PathLoc of clinical isolates does not predict increased biological activity of Toxin B or Toxin AA mariner-based transposon system for in vivo random mutagenesis of Clostridium difficile.Clostridium difficile extracytoplasmic function σ factor σV regulates lysozyme resistance and is necessary for pathogenesis in the hamster model of infection.Utility of the clostridial site-specific recombinase TnpX to clone toxic-product-encoding genes and selectively remove genomic DNA fragments.Identification and characterization of a gene cluster required for proper rod shape, cell division, and pathogenesis in Clostridium difficile.Group II intron-anchored gene deletion in ClostridiumThe Clostridium difficile proline racemase is not essential for early logarithmic growth and infection.The VirSR two-component signal transduction system regulates NetB toxin production in Clostridium perfringens.The cysteine protease α-clostripain is not essential for the pathogenesis of Clostridium perfringens-mediated myonecrosis.The role of toxin A and toxin B in Clostridium difficile-associated disease: Past and present perspectives.The anti-sigma factor TcdC modulates hypervirulence in an epidemic BI/NAP1/027 clinical isolate of Clostridium difficile.Pleiotropic role of the RNA chaperone protein Hfq in the human pathogen Clostridium difficileSecretion of Clostridium difficile toxins A and B requires the holin-like protein TcdE.Temporal differential proteomes of Clostridium difficile in the pig ileal-ligated loop modelIdentification of a genetic locus responsible for antimicrobial peptide resistance in Clostridium difficileExpanding the repertoire of gene tools for precise manipulation of the Clostridium difficile genome: allelic exchange using pyrE alleles.Genome-wide identification of regulatory RNAs in the human pathogen Clostridium difficile.TcsL is an essential virulence factor in Clostridium sordellii ATCC 9714Global analysis of the sporulation pathway of Clostridium difficile.Regulation of sialidase production in Clostridium perfringens by the orphan sensor histidine kinase ReeSSpo0A differentially regulates toxin production in evolutionarily diverse strains of Clostridium difficileUse of mCherry Red fluorescent protein for studies of protein localization and gene expression in Clostridium difficileRegulation of virulence by the RevR response regulator in Clostridium perfringens.Two novel membrane proteins, TcpD and TcpE, are essential for conjugative transfer of pCW3 in Clostridium perfringens.The dlt operon confers resistance to cationic antimicrobial peptides in Clostridium difficilePrsW is required for colonization, resistance to antimicrobial peptides, and expression of extracytoplasmic function σ factors in Clostridium difficile.Necrotic enteritis-derived Clostridium perfringens strain with three closely related independently conjugative toxin and antibiotic resistance plasmids.An alkaline phosphatase reporter for use in Clostridium difficile.Complete genome sequence of the Clostridium difficile laboratory strain 630Δerm reveals differences from strain 630, including translocation of the mobile element CTn5.
P2860
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P2860
Construction and analysis of chromosomal Clostridium difficile mutants.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Construction and analysis of chromosomal Clostridium difficile mutants.
@en
type
label
Construction and analysis of chromosomal Clostridium difficile mutants.
@en
prefLabel
Construction and analysis of chromosomal Clostridium difficile mutants.
@en
P2093
P2860
P50
P1476
Construction and analysis of chromosomal Clostridium difficile mutants.
@en
P2093
Jackie K Cheung
Jennifer R O'Connor
Kylie A Farrow
Vicki Adams
P2860
P304
P356
10.1111/J.1365-2958.2006.05315.X
P407
P577
2006-09-01T00:00:00Z