Activation of the spinal ERK signaling pathway contributes naloxone-precipitated withdrawal in morphine-dependent rats.
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The mechanism of μ-opioid receptor (MOR)-TRPV1 crosstalk in TRPV1 activation involves morphine anti-nociception, tolerance and dependenceLPS-induced TNF-alpha factor (LITAF)-deficient mice express reduced LPS-induced cytokine: Evidence for LITAF-dependent LPS signaling pathwaysFrequency-dependent ERK phosphorylation in spinal neurons by electric stimulation of the sciatic nerve and the role in electrophysiological activitySpinal mediators that may contribute selectively to antinociceptive tolerance but not other effects of morphine as revealed by deletion of GluR5Distribution of TRPC6 in the cerebrospinal fluid-contacting nucleus of rat brain parenchyma and its expression in morphine dependence and withdrawal.Mu opioid signaling protects against acute murine intestinal injury in a manner involving Stat3 signalingPhosphatidylethanolamine-binding protein is not involved in µ-opioid receptor-mediated regulation of extracellular signal-regulated kinase.Morphine withdrawal stress modulates lipopolysaccharide-induced interleukin 12 p40 (IL-12p40) expression by activating extracellular signal-regulated kinase 1/2, which is further potentiated by glucocorticoidsExtracellular signal-regulated kinase activation in the amygdala mediates elevated plus maze behavior during opioid withdrawalRSK2 signaling in medial habenula contributes to acute morphine analgesia.Opioid receptor trafficking and signaling: what happens after opioid receptor activation?Morphine Reduces Expression of TRPV1 Receptors in the Amygdala but not in the Hippocampus of Male Rats.Intrathecal injection of GluR6 antisense oligodeoxynucleotides alleviates acute inflammatory pain of rectum in rats.Activation of the ERK signalling pathway contributes to the adaptive changes in rat hearts during naloxone-induced morphine withdrawalThe PKs PKA and ERK 1/2 are involved in phosphorylation of TH at Serine 40 and 31 during morphine withdrawal in rat hearts.Transgene-mediated enkephalin expression attenuates signs of naloxone-precipitated morphine withdrawal in rats with neuropathic pain.The role of TNFα in the periaqueductal gray during naloxone-precipitated morphine withdrawal in ratsThe effects of morphine treatment on the NCAM and its signaling in the MLDS of rats.Extracellular signal-regulated kinase 5 in the cerebrospinal fluid-contacting nucleus contributes to morphine physical dependence in rats.Glucocorticoid receptor but not mineralocorticoid receptor mediates the activation of ERK pathway and CREB during morphine withdrawal.An emerging new paradigm in opioid withdrawal: a critical role for glia-neuron signaling in the periaqueductal gray.
P2860
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P2860
Activation of the spinal ERK signaling pathway contributes naloxone-precipitated withdrawal in morphine-dependent rats.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年學術文章
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2005年學術文章
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2005年學術文章
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name
Activation of the spinal ERK s ...... al in morphine-dependent rats.
@en
type
label
Activation of the spinal ERK s ...... al in morphine-dependent rats.
@en
prefLabel
Activation of the spinal ERK s ...... al in morphine-dependent rats.
@en
P2093
P1433
P1476
Activation of the spinal ERK s ...... al in morphine-dependent rats.
@en
P2093
Hai-Lei Ding
Jian-Hua He
Jun-Li Cao
Yin-Ming Zeng
P304
P356
10.1016/J.PAIN.2005.09.006
P407
P577
2005-11-14T00:00:00Z