about
A novel lipopolysaccharide-induced transcription factor regulating tumor necrosis factor alpha gene expression: molecular cloning, sequencing, characterization, and chromosomal assignmentSecreted frizzled-related protein 1 (SFRP1) protects fibroblasts from ceramide-induced apoptosisLPS induces the interaction of a transcription factor, LPS-induced TNF-alpha factor, and STAT6(B) with effects on multiple cytokinesToll-like receptor-2 mediates diet and/or pathogen associated atherosclerosis: proteomic findingsA new transcription factor that regulates TNF-alpha gene expression, LITAF, is increased in intestinal tissues from patients with CD and UCLPS-induced TNF-alpha factor (LITAF)-deficient mice express reduced LPS-induced cytokine: Evidence for LITAF-dependent LPS signaling pathwaysWhole-body deletion of LPS-induced TNF-α factor (LITAF) markedly improves experimental endotoxic shock and inflammatory arthritisLITAF and TNFSF15, two downstream targets of AMPK, exert inhibitory effects on tumor growthIdentification and functional characterization of a novel binding site on TNF-alpha promoter.Animal models for Porphyromonas gingivalis-mediated periodontal disease.Identification and characterization of kava-derived compounds mediating TNF-alpha suppression.Amelioration of emphysema in mice through lentiviral transduction of long-lived pulmonary alveolar macrophages.Bioinformatics analysis of macrophages exposed to Porphyromonas gingivalis: implications in acute vs. chronic infections.Cytokine profiling of macrophages exposed to Porphyromonas gingivalis, its lipopolysaccharide, or its FimA protein.LITAF, a BCL6 target gene, regulates autophagy in mature B-cell lymphomas.Novel regulation of CCL2 gene expression by murine LITAF and STAT6B.LITAF mediation of increased TNF-α secretion from inflamed colonic lamina propria macrophages.Periodontal innate immune mechanisms relevant to atherosclerosis and obesity.Contribution of interleukin-11 and prostaglandin(s) in lipopolysaccharide-induced bone resorption in vivo.A PTP4A3 peptide PIMAP39 modulates TNF-alpha levels and endotoxic shock.Controlling the outcome of the Toll-like receptor signaling pathways.Deep sequencing of the oral microbiome reveals signatures of periodontal disease.Signaling mechanisms in the restoration of impaired immune function due to diet-induced obesityPartial restoration of macrophage alteration from diet-induced obesity in response to Porphyromonas gingivalis infection.Periodontal innate immune mechanisms relevant to atherosclerosis.Diet-induced obesity in mice causes changes in immune responses and bone loss manifested by bacterial challenge.Periodontal disease and systemic conditions: a bidirectional relationship.Metabolic network model of a human oral pathogen.Macrophage polarization: an opportunity for improved outcomes in biomaterials and regenerative medicineKavain Inhibition of LPS-Induced TNF-α via ERK/LITAF.Convergent Synthesis of Novel Muramyl Dipeptide Analogues: Inhibition of Porphyromonas gingivalis-Induced Pro-inflammatory Effects by High Doses of Muramyl Dipeptide.Differentiation of monocytes to macrophages primes cells for lipopolysaccharide stimulation via accumulation of cytoplasmic nuclear factor kappaB.Moesin functions as a lipopolysaccharide receptor on human monocytes.Moesin-induced signaling in response to lipopolysaccharide in macrophages.Is Porphyromonas gingivalis cell invasion required for atherogenesis? Pharmacotherapeutic implications.p53 short peptide (p53pep164) regulates lipopolysaccharide-induced tumor necrosis factor-alpha factor/cytokine expression.Role of interleukin-1 in bacterial atherogenesis.Beneficial dysregulation of the time course of inflammatory mediators in lipopolysaccharide-induced tumor necrosis factor alpha factor-deficient mice.Identification and Characterization of MicroRNA Differentially Expressed in Macrophages Exposed to Porphyromonas gingivalis Infection.Molecular and cellular biology research in periodontal regeneration and its clinical implication.
P50
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Salomon Amar
@ast
Salomon Amar
@en
Salomon Amar
@es
Salomon Amar
@nl
Salomon Amar
@sl
type
label
Salomon Amar
@ast
Salomon Amar
@en
Salomon Amar
@es
Salomon Amar
@nl
Salomon Amar
@sl
prefLabel
Salomon Amar
@ast
Salomon Amar
@en
Salomon Amar
@es
Salomon Amar
@nl
Salomon Amar
@sl
P106
P21
P31
P496
0000-0002-0017-5930