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Striatal neuroinflammation promotes Parkinsonism in ratsChanges of hippocampal Cu/Zn-superoxide dismutase after kainate treatment in the ratEndogenous dynorphin protects against neurotoxin-elicited nigrostriatal dopaminergic neuron damage and motor deficits in miceCloning and expression of MP13 gene from rat hippocampus, a new factor related to guanosine triphosphate regulation.Glial cell line-derived neurotrophic factor protects midbrain dopaminergic neurons against lipopolysaccharide neurotoxicity.Expression of microsomal epoxide hydrolase is elevated in Alzheimer's hippocampus and induced by exogenous beta-amyloid and trimethyl-tin.Cyclooxygenase-2 mediates microglial activation and secondary dopaminergic cell death in the mouse MPTP model of Parkinson's disease.Dextromethorphan attenuates trimethyltin-induced neurotoxicity via sigma1 receptor activation in rats.Role of microsomal epoxide hydrolase in methamphetamine-induced drug dependence in mice.Studies on the cellular localization of biochemical responses to catecholamines in the brain.Traumatic brain injury and trichloroethylene exposure interact and produce functional, histological, and mitochondrial deficitsPioglitazone inhibition of lipopolysaccharide-induced nitric oxide synthase is associated with altered activity of p38 MAP kinase and PI3K/AktPKCĪ“ inhibition enhances tyrosine hydroxylase phosphorylation in mice after methamphetamine treatment.Lipopolysaccharide animal models for Parkinson's disease.Long-term increase of Sp-1 transcription factors in the hippocampus after kainic acid treatment.Adrenal chromaffin cells as transplants in animal models of Parkinson's disease.PINK1/Parkin-mediated mitophagy alleviates chlorpyrifos-induced apoptosis in SH-SY5Y cells.Striatal GDNF administration increases tyrosine hydroxylase phosphorylation in the rat striatum and substantia nigra.Protective properties afforded by pioglitazone against intrastriatal LPS in Sprague-Dawley rats.Pioglitazone attenuates mitochondrial dysfunction, cognitive impairment, cortical tissue loss, and inflammation following traumatic brain injury.Selective killing of cholinergic neurons by microglial activation in basal forebrain mixed neuronal/glial cultures.The effect of HMGB1 on sub-toxic chlorpyrifos exposure-induced neuroinflammation in amygdala of neonatal rats.Neonatal chlorpyrifos exposure induces loss of dopaminergic neurons in young adult rats.Interleukin-10 protects against inflammation-mediated degeneration of dopaminergic neurons in substantia nigra.Potentiation of methamphetamine neurotoxicity by intrastriatal lipopolysaccharide administration.Induction of unspliced c-fos messenger RNA in rodent brain by kainic acid and lipopolysaccharide.Dextromethorphan affects cocaine-mediated behavioral pattern in parallel with a long-lasting Fos-related antigen-immunoreactivity.Prenatal exposure to magnetic field increases dopamine levels in the striatum of offspring.Oxidative damage causes formation of lipofuscin-like substances in the hippocampus of the senescence-accelerated mouse after kainate treatment.Localization of beta-secretase memapsin 2 in the brain of Alzheimer's patients and normal aged controls.Metabolism to dextrorphan is not essential for dextromethorphan's anticonvulsant activity against kainate in mice.Kainate treatment alters TGF-beta3 gene expression in the rat hippocampus.Up-regulation of inducible nitric oxide synthase in the substantia nigra by lipopolysaccharide causes microglial activation and neurodegeneration.Dextromethorphan blocks opioid peptide gene expression in the rat hippocampus induced by kainic acid.Noradrenergic activation of immediate early genes in rat cerebral cortex.Role of the prolyl isomerase Pin1 in protecting against age-dependent neurodegeneration.c-Fos response to administration of catecholamines into brain by microdialysis.Prodynorphin gene deficiency potentiates nalbuphine-induced behavioral sensitization and withdrawal syndrome in mice.Effects of prodynorphin deletion on striatal dopamine in mice during normal aging and in response to MPTP.Phenidone protects the nigral dopaminergic neurons from LPS-induced neurotoxicity.
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