Inducible nitric-oxide synthase and NO donor induce insulin receptor substrate-1 degradation in skeletal muscle cells.
about
Regulation of obesity and insulin resistance by nitric oxideThe UPS in diabetes and obesity.Deletion of inducible nitric-oxide synthase in leptin-deficient mice improves brown adipose tissue function.Farnesyltransferase inhibitor improved survival following endotoxin challenge in mice.AMP-activated protein kinase (AMPK)α2 plays a role in determining the cellular fate of glucose in insulin-resistant mouse skeletal muscle.iNOS inhibitor, L-NIL, reverses burn-induced glycogen synthase kinase-3β activation in skeletal muscle of rats.Inducible nitric oxide synthase deficiency in myeloid cells does not prevent diet-induced insulin resistanceCXCR3 modulates obesity-induced visceral adipose inflammation and systemic insulin resistance.Protein S-nitrosylation in health and disease: a current perspective.Inflammatory stimuli induce inhibitory S-nitrosylation of the deacetylase SIRT1 to increase acetylation and activation of p53 and p65Increased adipocyte S-nitrosylation targets anti-lipolytic action of insulin: relevance to adipose tissue dysfunction in obesityLiver-specific inducible nitric-oxide synthase expression is sufficient to cause hepatic insulin resistance and mild hyperglycemia in mice.Ethyl pyruvate preserves IGF-I sensitivity toward mTOR substrates and protein synthesis in C2C12 myotubes.Farnesyltransferase inhibitor FTI-277 reduces mortality of septic mice along with improved bacterial clearanceVascular smooth muscle insulin resistance, but not hypertrophic signaling, is independent of angiotensin II-induced IRS-1 phosphorylation by JNKEndotoxin-Binding Peptides Derived from Casein Glycomacropeptide Inhibit Lipopolysaccharide-Stimulated Inflammatory Responses via Blockade of NF-κB activation in macrophagesInducible nitric oxide synthase deficiency ameliorates skeletal muscle insulin resistance but does not alter unexpected lower blood glucose levels after burn injury in C57BL/6 miceLuteolin is a bioflavonoid that attenuates adipocyte-derived inflammatory responses via suppression of nuclear factor-κB/mitogen-activated protein kinases pathway.iNOS as a Driver of Inflammation and Apoptosis in Mouse Skeletal Muscle after Burn Injury: Possible Involvement of Sirt1 S-Nitrosylation-Mediated Acetylation of p65 NF-κB and p53Obesity, insulin resistance, and skeletal muscle nitric oxide synthaseTargeted disruption of inducible nitric oxide synthase protects against aging, S-nitrosation, and insulin resistance in muscle of male miceObese mice lacking inducible nitric oxide synthase are sensitized to the metabolic actions of peroxisome proliferator-activated receptor-gamma agonism.Skeletal muscle insulin resistance is fundamental to the cardiometabolic syndrome.Rapamycin does not improve insulin sensitivity despite elevated mammalian target of rapamycin complex 1 activity in muscles of ob/ob mice.Exercise training improves obesity-related lymphatic dysfunctionDual mechanisms of CYP3A protein regulation by proinflammatory cytokine stimulation in primary hepatocyte cultures.Obesity-induced insulin resistance and hyperglycemia: etiologic factors and molecular mechanisms.Insulin resistance postburn: underlying mechanisms and current therapeutic strategies.Nitrosative modifications of protein and lipid signaling molecules by reactive nitrogen species.Regulation of pancreatic β-cell survival by nitric oxide: clinical relevance.Targeted disruption of iNOS prevents LPS-induced S-nitrosation of IRbeta/IRS-1 and Akt and insulin resistance in muscle of mice.Transcriptional analysis of brown adipose tissue in leptin-deficient mice lacking inducible nitric oxide synthase: evidence of the role of Med1 in energy balance.Emerging Role of Nitric Oxide and Heat Shock Proteins in Insulin Resistance.Nitric oxide and AMPK cooperatively regulate PGC-1 in skeletal muscle cells.Effect of insulin levels on the phosphorylation of specific amino acid residues in IRS-1: implications for burn-induced insulin resistance.Nitric oxide facilitates NFAT-dependent transcription in mouse myotubes.ERK5 activation inhibits inflammatory responses via peroxisome proliferator-activated receptor delta (PPARdelta) stimulation.Role of protein farnesylation in burn-induced metabolic derangements and insulin resistance in mouse skeletal muscle.Altered glucose and lipid homeostasis in liver and adipose tissue pre-dispose inducible NOS knockout mice to insulin resistance.Brussels 2007 roundtable on metabolism in sepsis and multiple organ failure.
P2860
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P2860
Inducible nitric-oxide synthase and NO donor induce insulin receptor substrate-1 degradation in skeletal muscle cells.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Inducible nitric-oxide synthas ...... tion in skeletal muscle cells.
@en
type
label
Inducible nitric-oxide synthas ...... tion in skeletal muscle cells.
@en
prefLabel
Inducible nitric-oxide synthas ...... tion in skeletal muscle cells.
@en
P2093
P2860
P356
P1476
Inducible nitric-oxide synthas ...... tion in skeletal muscle cells.
@en
P2093
Hiroki Sugita
J A Jeevendra Martyn
Masaki Fujimoto
Masao Kaneki
Michiko Sugita
Nobuyuki Shimizu
Shingo Yasuhara
Takashi Yasukawa
P2860
P304
14203-14211
P356
10.1074/JBC.M411226200
P407
P577
2005-02-01T00:00:00Z