about
Anti-inflammatory dimethylfumarate: a potential new therapy for asthma?Ligand-independent activation of the glucocorticoid receptor by beta2-adrenergic receptor agonists in primary human lung fibroblasts and vascular smooth muscle cellsCa2+ channel blockers modulate metabolism of collagens within the extracellular matrixTime-dependent and somatically acquired mitochondrial DNA mutagenesis and respiratory chain dysfunction in a scleroderma model of lung fibrosisThe role of IgE-receptors in IgE-dependent airway smooth muscle cell remodellingPrimary fibroblast cell cultures from transbronchial biopsies of lung transplant recipients.Intracellular interleukin 6 mediates platelet-derived growth factor-induced proliferation of nontransformed cells.Increased proinflammatory responses from asthmatic human airway smooth muscle cells in response to rhinovirus infectionA granulocyte inhibitory protein overexpressed in chronic renal disease regulates expression of interleukin 6 and interleukin 8.The interaction of endothelin-1 and TGF-β1 mediates vascular cell remodeling.Bronchial smooth muscle cells of asthmatics promote angiogenesis through elevated secretion of CXC-chemokines (ENA-78, GRO-α, and IL-8).Chlamydia pneumoniae infections in asthma: clinical implications.Serum IgE Induced Airway Smooth Muscle Cell Remodeling Is Independent of Allergens and Is Prevented by OmalizumabCalreticulin is a negative regulator of bronchial smooth muscle cell proliferationAcute exacerbations of COPD are associated with significant activation of matrix metalloproteinase 9 irrespectively of airway obstruction, emphysema and infection.Asthma and COPD - The C/EBP ConnectionComparison of gel contraction mediated by airway smooth muscle cells from patients with and without asthma.Transcriptional activation of low density lipoprotein receptor gene by angiotensin-converting enzyme inhibitors and Ca(2+)-channel blockers involves protein kinase C isoformsTranscription factors in asthma: are transcription factors a new target for asthma therapy?Mannose-binding lectin protein and its association to clinical outcomes in COPD: a longitudinal study.An imbalance in C/EBPs and increased mitochondrial activity in asthmatic airway smooth muscle cells: novel targets in asthma therapy?Intrinsic asthma: is it intrinsic to the smooth muscle?Ca(2+)-channel blockers modulate expression of 3-hydroxy-3-methylglutaryl-coenzyme A reductase and low density lipoprotein receptor genes stimulated by platelet-derived growth factorMolecular mechanisms of combination therapy with inhaled corticosteroids and long-acting beta-agonists.Human airway smooth muscle cell proliferation from asthmatics is negatively regulated by semaphorin3A.Airway smooth muscle cells respond directly to inhaled environmental factors.Chlamydia pneumoniae infection after lung transplantation.Chlamydia pneumoniae activates epithelial cell proliferation via NF-kappaB and the glucocorticoid receptor.Lung remodeling mechanisms in chronic lung diseases.Clinical potential of aclidinium bromide in chronic obstructive pulmonary diseaseAirway and lung remodelling in chronic pulmonary obstructive disease: a role for muscarinic receptor antagonists?Transforming growth factor-beta1 induces interleukin-6 expression via activating protein-1 consisting of JunD homodimers in primary human lung fibroblasts.Dysfunctional interaction of C/EBPalpha and the glucocorticoid receptor in asthmatic bronchial smooth-muscle cells.Specific regulation of PRMT1 expression by PIAS1 and RKIP in BEAS-2B epithelia cells and HFL-1 fibroblasts in lung inflammation.PDGF-BB induces PRMT1 expression through ERK1/2 dependent STAT1 activation and regulates remodeling in primary human lung fibroblasts.Hepatocyte growth factor secreted by bone marrow stem cell reduce ER stress and improves repair in alveolar epithelial II cells.Hyaluronic acid: A key molecule in skin aging.The MNK-1/eIF4E pathway as a new therapeutic pathway to target inflammation and remodelling in asthma.Rat mesothelioma cell proliferation requires p38δ mitogen activated protein kinase and C/EBP-α.Mesothelioma cells escape heat stress by upregulating Hsp40/Hsp70 expression via mitogen-activated protein kinases.
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description
hulumtues
@sq
onderzoeker
@nl
researcher
@en
ricercatore
@it
հետազոտող
@hy
name
Michael Roth
@ast
Michael Roth
@de
Michael Roth
@en
Michael Roth
@es
Michael Roth
@sl
type
label
Michael Roth
@ast
Michael Roth
@de
Michael Roth
@en
Michael Roth
@es
Michael Roth
@sl
altLabel
M Roth
@en
prefLabel
Michael Roth
@ast
Michael Roth
@de
Michael Roth
@en
Michael Roth
@es
Michael Roth
@sl
P214
P227
P106
P21
P214
P227
1090345623
P31
P496
0000-0002-8139-2821
P5008
P569
1955-01-01T00:00:00Z
P734
P735
P7859
lccn-n85249142