Rolling of human bone-metastatic prostate tumor cells on human bone marrow endothelium under shear flow is mediated by E-selectin.
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Identification of leukocyte E-selectin ligands, P-selectin glycoprotein ligand-1 and E-selectin ligand-1, on human metastatic prostate tumor cellsTargeting selectins and selectin ligands in inflammation and cancerLeveraging fluorinated glucosamine action to boost antitumor immunityCirculating tumor cells from prostate cancer patients interact with E-selectin under physiologic blood flowCD44 variant isoforms expressed by breast cancer cells are functional E-selectin ligands under flow conditionsP-selectin mediates metastatic progression through binding to sulfatides on tumor cellsMac-2 binding protein is a novel E-selectin ligand expressed by breast cancer cellsHigh Throughput Label Free Measurement of Cancer Cell Adhesion Kinetics Under Hemodynamic Flow.Alpha 1,3 fucosyltransferases are master regulators of prostate cancer cell trafficking.Knockdown of fucosyltransferase III disrupts the adhesion of circulating cancer cells to E-selectin without affecting hematopoietic cell adhesionShear flow-induced formation of tubular cell protrusions in multiple myeloma cells.E-selectin liposomal and nanotube-targeted delivery of doxorubicin to circulating tumor cells.High-content adhesion assay to address limited cell samplesHuman fucosyltransferase 6 enables prostate cancer metastasis to boneHuman antibodies targeting cell surface antigens overexpressed by the hormone refractory metastatic prostate cancer cells: ICAM-1 is a tumor antigen that mediates prostate cancer cell invasion.Endothelial integrin α3β1 stabilizes carbohydrate-mediated tumor/endothelial cell adhesion and induces macromolecular signaling complex formation at the endothelial cell membrane.Skin-homing receptors on effector leukocytes are differentially sensitive to glyco-metabolic antagonism in allergic contact dermatitis.Enhanced tumor cell isolation by a biomimetic combination of E-selectin and anti-EpCAM: implications for the effective separation of circulating tumor cells (CTCs)In vitro method to observe E-selectin-mediated interactions between prostate circulating tumor cells derived from patients and human endothelial cellsDirect measurements on CD24-mediated rolling of human breast cancer MCF-7 cells on E-selectin.Suppression of prostate cancer cell rolling and adhesion to endothelium by 1α,25-dihydroxyvitamin D3.Peracetylated 4-fluoro-glucosamine reduces the content and repertoire of N- and O-glycans without direct incorporation.A high-throughput mechanofluidic screening platform for investigating tumor cell adhesion during metastasis.Glycosyltransferases involved in the synthesis of MUC-associated metastasis-promoting selectin ligands.Effects of Panax notoginseng on the Metastasis of Human Colorectal Cancer Cells.Expression of E-selectin ligands on circulating tumor cells: cross-regulation with cancer stem cell regulatory pathways?Dendrimer-mediated multivalent binding for the enhanced capture of tumor cells.Analysis of glycosyltransferase expression in metastatic prostate cancer cells capable of rolling activity on microvascular endothelial (E)-selectin.Alterations in human breast cancer adhesion-motility in response to changes in cell surface glycoproteins displaying alpha-L-fucose moieties.Analysis of physiologic E-selectin-mediated leukocyte rolling on microvascular endothelium.Physical biology in cancer. 3. The role of cell glycocalyx in vascular transport of circulating tumor cells.Leukocytes as carriers for targeted cancer drug delivery.Galectin-3 expressed on different lung compartments promotes organ specific metastasis by facilitating arrest, extravasation and organ colonization via high affinity ligands on melanoma cells.Assessment of Molecular Acoustic Angiography for Combined Microvascular and Molecular Imaging in Preclinical Tumor Models.C-type lectins facilitate tumor metastasis.Definition of molecular determinants of prostate cancer cell bone extravasation.The interaction between CD44 on tumour cells and hyaluronan under physiologic flow conditions: implications for metastasis formation.Gene network and canonical pathway analysis in prostate cancer: a microarray study.The p38 pathway, a major pleiotropic cascade that transduces stress and metastatic signals in endothelial cells.Proangiogenic stimulation of bone marrow endothelium engages mTOR and is inhibited by simultaneous blockade of mTOR and NF-kappaB.
P2860
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P2860
Rolling of human bone-metastatic prostate tumor cells on human bone marrow endothelium under shear flow is mediated by E-selectin.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Rolling of human bone-metastat ...... low is mediated by E-selectin.
@en
type
label
Rolling of human bone-metastat ...... low is mediated by E-selectin.
@en
prefLabel
Rolling of human bone-metastat ...... low is mediated by E-selectin.
@en
P2093
P1433
P1476
Rolling of human bone-metastat ...... low is mediated by E-selectin.
@en
P2093
Charles J Dimitroff
Denise Long-Woodward
Jeffery L Kutok
Mirna Lechpammer
P304
P356
10.1158/0008-5472.CAN-04-0691
P407
P577
2004-08-01T00:00:00Z