Cytokines increase CRE binding but decrease CRE-mediated reporter activity in rat hepatocytes by increasing c-Jun.
about
Nonclassical action of retinoic acid on the activation of the cAMP response element-binding protein in normal human bronchial epithelial cellsAkt-mediated signaling is induced by cytokines and cyclic adenosine monophosphate and suppresses hepatocyte inducible nitric oxide synthase expression independent of MAPK P44/42.Functional repression of cAMP response element in 6-hydroxydopamine-treated neuronal cells.Activation of a cyclic amp-guanine exchange factor in hepatocytes decreases nitric oxide synthase expression.Aryl hydrocarbon receptor activation attenuates Per1 gene induction and influences circadian clock resetting.Restoration of CREB function ameliorates cisplatin cytotoxicity in renal tubular cells.CREB/PKA sensitive signalling pathways activate and maintain expression levels of the hepatitis B virus pre-S2/S promoter.Retinoic acid induces caspase-8 transcription via phospho-CREB and increases apoptotic responses to death stimuli in neuroblastoma cells.Glycogen synthase kinase 3 regulates IL-1β mediated iNOS expression in hepatocytes by down-regulating c-Jun.
P2860
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P2860
Cytokines increase CRE binding but decrease CRE-mediated reporter activity in rat hepatocytes by increasing c-Jun.
description
2004 nî lūn-bûn
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name
Cytokines increase CRE binding ...... patocytes by increasing c-Jun.
@en
type
label
Cytokines increase CRE binding ...... patocytes by increasing c-Jun.
@en
prefLabel
Cytokines increase CRE binding ...... patocytes by increasing c-Jun.
@en
P2093
P2860
P356
P1433
P1476
Cytokines increase CRE binding ...... patocytes by increasing c-Jun.
@en
P2093
Baochun Zhang
Brian G Harbrecht
Michele D Perpetua
Shubing Liu
William H Walker
P2860
P304
P356
10.1002/HEP.20200
P407
P577
2004-05-01T00:00:00Z