Ablation of C/EBP homologous protein attenuates endoplasmic reticulum-mediated apoptosis and cardiac dysfunction induced by pressure overload.
about
The Role of Endoplasmic Reticulum Stress and Unfolded Protein Response in AtherosclerosisRole for the Unfolded Protein Response in Heart Disease and Cardiac ArrhythmiasCell death and survival through the endoplasmic reticulum-mitochondrial axisKeep your heart in shape: molecular chaperone networks for treating heart diseaseAblation of C/EBP homologous protein does not protect T17M RHO mice from retinal degenerationOxidative stress, unfolded protein response, and apoptosis in developmental toxicityProtein quality control and metabolism: bidirectional control in the heartTargeted Therapy for Acute Autoimmune Myocarditis with Nano-Sized Liposomal FK506 in RatsIntegrating the mechanisms of apoptosis induced by endoplasmic reticulum stressPrenatal exposure to lipopolysaccharide results in myocardial remodelling in adult murine offspring.Tauroursodeoxycholic acid (TUDCA) attenuates pressure overload-induced cardiac remodeling by reducing endoplasmic reticulum stress.Heavy metal scavenger metallothionein attenuates ER stress-induced myocardial contractile anomalies: role of autophagyParkin regulation of CHOP modulates susceptibility to cardiac endoplasmic reticulum stress.Macrophage Apoptosis and Efferocytosis in the Pathogenesis of AtherosclerosisThrombin increases lung fibroblast survival while promoting alveolar epithelial cell apoptosis via the endoplasmic reticulum stress marker, CCAAT enhancer-binding homologous proteinEndoplasmic reticulum stress sensor protein kinase R-like endoplasmic reticulum kinase (PERK) protects against pressure overload-induced heart failure and lung remodeling.Induction of apoptosis coupled to endoplasmic reticulum stress in human prostate cancer cells by n-butylidenephthalide.Increased expression of fatty-acid and calcium metabolism genes in failing human heart.Activation of volume-sensitive outwardly rectifying chloride channel by ROS contributes to ER stress and cardiac contractile dysfunction: involvement of CHOP through Wnt.Klotho suppresses cardiomyocyte apoptosis in mice with stress-induced cardiac injury via downregulation of endoplasmic reticulum stress.The sarcoplasmic reticulum luminal thiol oxidase ERO1 regulates cardiomyocyte excitation-coupled calcium release and response to hemodynamic load.Guanabenz interferes with ER stress and exerts protective effects in cardiac myocytes.Cell death signalling mechanisms in heart failure.Decreased metalloprotease 9 induction, cardiac fibrosis, and higher autophagy after pressure overload in mice lacking the transcriptional regulator p8.Cardiomyocyte death: mechanisms and translational implicationsInflammation, endoplasmic reticulum stress, autophagy, and the monocyte chemoattractant protein-1/CCR2 pathway.Hydrogen sulfide preconditioning protects against myocardial ischemia/reperfusion injury in rats through inhibition of endo/sarcoplasmic reticulum stressEndoplasmic Reticulum Stress and Unfolded Protein Response Pathways: Potential for Treating Age-related Retinal DegenerationDeficiency of αB crystallin augments ER stress-induced apoptosis by enhancing mitochondrial dysfunctionMicroRNA regulation of unfolded protein response transcription factor XBP1 in the progression of cardiac hypertrophy and heart failure in vivo.Bushen Zhuangjin decoction inhibits TM-induced chondrocyte apoptosis mediated by endoplasmic reticulum stress.Modulation of the unfolded protein response during hepatocyte and cardiomyocyte apoptosis in trauma/hemorrhagic shockIcariin protects rat cardiac H9c2 cells from apoptosis by inhibiting endoplasmic reticulum stressHistone Deacetylase Inhibitor Phenylbutyrate Exaggerates Heart Failure in Pressure Overloaded Mice independently of HDAC inhibition.Loss of the eukaryotic initiation factor 2α kinase general control nonderepressible 2 protects mice from pressure overload-induced congestive heart failure without affecting ventricular hypertrophy.CYP2J2-derived epoxyeicosatrienoic acids suppress endoplasmic reticulum stress in heart failure.Cardiac aging and insulin resistance: could insulin/insulin-like growth factor (IGF) signaling be used as a therapeutic target?Endoplasmic reticulum stress in bone marrow-derived cells prevents acute cardiac inflammation and injury in response to angiotensin II.Unfolded protein response signaling and metabolic diseases.A modified approach to induce predictable congestive heart failure by volume overload in rats.
P2860
Q26769783-261B5C56-5738-45D8-8250-1B3C1CDC7245Q26771256-145AF6D9-E2C2-4095-8F15-D3F309A0BC74Q26823387-4A93C1B7-6FDF-4105-9B83-B2E84D72B9D4Q27010429-4E8C7803-E1C2-4C45-8344-5DE28A79FD54Q27321032-38AF13EA-9527-4B0C-AF9F-AE9B1773C0D8Q28084613-6A3DEA92-4A2B-445D-BA48-2AA14C8AEB41Q28256472-3CB4F78F-DCAF-4F8F-B4A9-2B06B1B864B9Q28553409-93784522-3031-4CE9-9759-2E45042BF779Q29615495-B7D449F2-5C8E-4A3E-9445-F8E41B694F64Q30445274-5A4085CA-DCF6-4D5D-B2A1-AEB12482985BQ33585295-EBEB79B6-4D2C-400F-963D-17B7650F15BAQ33698356-CDC10726-9B5D-4E6F-BAC5-9624B601A303Q33704728-D9807E32-EEA6-49DC-8CE0-201D05D0FB05Q33764821-34B2B607-3ACF-4E1B-8333-4FBF029003BDQ33799719-C26D577C-0639-408C-B9FA-C45A687F1665Q34173536-9FC88B36-8239-4EB3-B991-F9A3C3BC781DQ34217464-9CE0C292-6746-49A6-8BF2-E519D37BCA28Q34305150-73C046FF-4333-4298-AC50-2EC25FFDBE3AQ34662249-F36CB60E-3440-4F36-B0C4-5DA4159C122EQ35067922-3382F09A-6C51-4C24-8B13-1248CE497154Q35105380-358B899A-347B-40B4-AA16-D147908680CEQ35180639-335287BD-49ED-4D1A-9BBE-27EC3C587160Q35500618-42EB3E2B-88F1-4472-BEEF-C1E438744CEAQ35543254-1040BDBD-F46C-4B5B-AC3E-B2AE19DED1E9Q35657122-A81ECB1C-42D9-4C2A-9129-60633E20B3DBQ35691449-403A373D-0F43-4DFD-8A8F-3FD20DDB2D0FQ36014153-FD943146-5613-40A5-AEA3-482A2834DC5AQ36052285-87C7B6B6-2980-4519-AE5F-55087C5E0C85Q36266703-BCB2B974-32E4-4610-852E-C9E05037B166Q36286532-7A5D90D6-98E3-497D-A5DC-404A933025CDQ36369257-BA58794C-7DFA-4F5C-9A71-6A822A5DA95FQ36579792-7F3E4668-AB37-4B04-9107-B3B2AEE7CE6FQ37225073-39C0E7E3-8372-4F30-8870-1301069FA5C8Q37281996-1BC1F57E-6B90-4C75-B868-A9E8E987A76FQ37365394-14499AED-9C98-46DB-B780-BB2F6AC05593Q37405408-5324700D-1B46-4352-8451-A9EEB1E9CD62Q37442678-F0B15469-5243-49FD-BB27-8E8BF66262BEQ37482793-226A9C5D-7E7C-44A9-942E-4E8967CF6FDBQ37488392-D6F6BC56-79E5-4CD7-AD3A-DE9106759115Q37538767-CF7FFE1F-60DD-497C-B01F-2BD4E347643F
P2860
Ablation of C/EBP homologous protein attenuates endoplasmic reticulum-mediated apoptosis and cardiac dysfunction induced by pressure overload.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
Ablation of C/EBP homologous p ...... induced by pressure overload.
@en
type
label
Ablation of C/EBP homologous p ...... induced by pressure overload.
@en
prefLabel
Ablation of C/EBP homologous p ...... induced by pressure overload.
@en
P2093
P1433
P1476
Ablation of C/EBP homologous p ...... induced by pressure overload.
@en
P2093
Hai Ying Fu
Hiroshi Asanuma
Keiji Okuda
Ken-ichiro Okada
Masafumi Kitakaze
Masanori Asakura
Mitsutoshi Asai
Osamu Tsukamoto
Seiji Takashima
Shoji Sanada
P304
P356
10.1161/CIRCULATIONAHA.109.917914
P407
P50
P577
2010-07-12T00:00:00Z