EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia. - Wikidata - awgldk - TriplyDB

EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia.

EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia.

http://www.wikidata.org/entity/Q38345216

about

Pediatric AML: From Biology to Clinical Management Evaluation of gene expression signatures predictive of cytogenetic and molecular subtypes of pediatric acute myeloid leukemia EVI1 inhibits apoptosis induced by antileukemic drugs via upregulation of CDKN1A/p21/WAF in human myeloid cells Global Identification of EVI1 Target Genes in Acute Myeloid Leukemia.Prognostic significance of additional cytogenetic aberrations in 733 de novo pediatric 11q23/MLL-rearranged AML patients: results of an international study Chromosomal aberrations associated with clonal evolution and leukemic transformation in fanconi anemia: clinical and biological implications.Collaborative Efforts Driving Progress in Pediatric Acute Myeloid Leukemia.EVI1 is critical for the pathogenesis of a subset of MLL-AF9-rearranged AMLs.High EVI1 expression is associated with MLL rearrangements and predicts decreased survival in paediatric acute myeloid leukaemia: a report from the children's oncology group.EVI1 expression in childhood acute lymphoblastic leukaemia is not restricted to MLL and BCR/ABL rearrangements and is influenced by age.Overexpression of Evi-1 oncoprotein represses TGF-β signaling in colorectal cancer.Molecular diagnosis of acute myeloid leukemia.The heterogeneity of pediatric MLL-rearranged acute myeloid leukemia.-7/7q- syndrome in myeloid-lineage hematopoietic malignancies: attempts to understand this complex disease entity.High expression of EVI1 and MEL1 is a compelling poor prognostic marker of pediatric AML.3q26/EVI1 rearrangements in myeloid hemopathies: a cytogenetic review.Review: Aberrant EVI1 expression in acute myeloid leukaemia.Aberrant expression of ecotropic viral integration site-1 in acute myeloid leukemia and acute lymphoblastic leukemia.Evi1 defines leukemia-initiating capacity and tyrosine kinase inhibitor resistance in chronic myeloid leukemia.A standardized microarray assay for the independent gene expression markers in AML: EVI1 and BAALC.EVI1 overexpression is a poor prognostic factor in pediatric patients with mixed lineage leukemia-AF9 rearranged acute myeloid leukemia.Response to lenalidomide of a patient with t(2;3)(p23;q29) and JAK2 non-mutated refractory anemia with ring sideroblasts and thrombocytosis.Clinical features and prognostic impact of PRDM16 expression in adult acute myeloid leukemia.Identification of novel MECOM gene fusion and personalized therapeutic targets through integrative clinical sequencing in secondary acute myeloid leukemia in a patient with severe congenital neutropenia: a case report and literature review.Ecotropic viral integration site 1 regulates the progression of acute myeloid leukemia via MS4A3-mediated TGFβ/EMT signaling pathway.High BRE expression in pediatric MLL-rearranged AML is associated with favorable outcome

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P2860

EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia.

http://www.wikidata.org/entity/Q38345216

description

2010 nî lūn-bûn

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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2010年论文

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2010年论文

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name

EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia.

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EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia.

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EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia.

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EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia.

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P2093

B V Balgobind

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C M Zwaan

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D Reinhardt

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E R van Wering

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E S J M de Bont

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G J L Kaspers

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H B Beverloo

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I H Hollink

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J Stary

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J Trka

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P2860

Intergenic splicing of MDS1 and EVI1 occurs in normal tissues as well as in myeloid leukemia and produces a new member of the PR domain family

Pbx1 is a downstream target of Evi-1 in hematopoietic stem/progenitors and leukemic cells

Activation of EVI1 gene expression in human acute myelogenous leukemias by translocations spanning 300-400 kilobases on chromosome band 3q26

Fusion with E2A converts the Pbx1 homeodomain protein into a constitutive transcriptional activator in human leukemias carrying the t(1;19) translocation

Activating mutation of D835 within the activation loop of FLT3 in human hematologic malignancies.

The World Health Organization (WHO) classification of the myeloid neoplasms

High EVI1 expression predicts poor survival in acute myeloid leukemia: a study of 319 de novo AML patients

EVI1 is expressed in megakaryocyte cell lineage and enforced expression of EVI1 in UT-7/GM cells induces megakaryocyte differentiation

Retroviral activation of a novel gene encoding a zinc finger protein in IL-3-dependent myeloid leukemia cell lines

EVI1 and hematopoietic disorders: history and perspectives

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24

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