EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia.
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Pediatric AML: From Biology to Clinical ManagementEvaluation of gene expression signatures predictive of cytogenetic and molecular subtypes of pediatric acute myeloid leukemiaEVI1 inhibits apoptosis induced by antileukemic drugs via upregulation of CDKN1A/p21/WAF in human myeloid cellsGlobal Identification of EVI1 Target Genes in Acute Myeloid Leukemia.Prognostic significance of additional cytogenetic aberrations in 733 de novo pediatric 11q23/MLL-rearranged AML patients: results of an international studyChromosomal aberrations associated with clonal evolution and leukemic transformation in fanconi anemia: clinical and biological implications.Collaborative Efforts Driving Progress in Pediatric Acute Myeloid Leukemia.EVI1 is critical for the pathogenesis of a subset of MLL-AF9-rearranged AMLs.High EVI1 expression is associated with MLL rearrangements and predicts decreased survival in paediatric acute myeloid leukaemia: a report from the children's oncology group.EVI1 expression in childhood acute lymphoblastic leukaemia is not restricted to MLL and BCR/ABL rearrangements and is influenced by age.Overexpression of Evi-1 oncoprotein represses TGF-β signaling in colorectal cancer.Molecular diagnosis of acute myeloid leukemia.The heterogeneity of pediatric MLL-rearranged acute myeloid leukemia.-7/7q- syndrome in myeloid-lineage hematopoietic malignancies: attempts to understand this complex disease entity.High expression of EVI1 and MEL1 is a compelling poor prognostic marker of pediatric AML.3q26/EVI1 rearrangements in myeloid hemopathies: a cytogenetic review.Review: Aberrant EVI1 expression in acute myeloid leukaemia.Aberrant expression of ecotropic viral integration site-1 in acute myeloid leukemia and acute lymphoblastic leukemia.Evi1 defines leukemia-initiating capacity and tyrosine kinase inhibitor resistance in chronic myeloid leukemia.A standardized microarray assay for the independent gene expression markers in AML: EVI1 and BAALC.EVI1 overexpression is a poor prognostic factor in pediatric patients with mixed lineage leukemia-AF9 rearranged acute myeloid leukemia.Response to lenalidomide of a patient with t(2;3)(p23;q29) and JAK2 non-mutated refractory anemia with ring sideroblasts and thrombocytosis.Clinical features and prognostic impact of PRDM16 expression in adult acute myeloid leukemia.Identification of novel MECOM gene fusion and personalized therapeutic targets through integrative clinical sequencing in secondary acute myeloid leukemia in a patient with severe congenital neutropenia: a case report and literature review.Ecotropic viral integration site 1 regulates the progression of acute myeloid leukemia via MS4A3-mediated TGFβ/EMT signaling pathway.High BRE expression in pediatric MLL-rearranged AML is associated with favorable outcome
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EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia.
@en
type
label
EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia.
@en
prefLabel
EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia.
@en
P2093
P2860
P356
P1433
P1476
EVI1 overexpression in distinct subtypes of pediatric acute myeloid leukemia.
@en
P2093
B V Balgobind
D Reinhardt
E R van Wering
E S J M de Bont
G J L Kaspers
H B Beverloo
I H Hollink
P2860
P2888
P304
P356
10.1038/LEU.2010.47
P577
2010-04-01T00:00:00Z
P5875
P6179
1047815691