Id-1 expression promotes cell survival through activation of NF-kappaB signalling pathway in prostate cancer cells. - Wikidata - awgldk - TriplyDB

Id-1 expression promotes cell survival through activation of NF-kappaB signalling pathway in prostate cancer cells.

Id-1 expression promotes cell survival through activation of NF-kappaB signalling pathway in prostate cancer cells.

http://www.wikidata.org/entity/Q38352246

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Identification of a novel inhibitor of differentiation-1 (ID-1) binding partner, caveolin-1, and its role in epithelial-mesenchymal transition and resistance to apoptosis in prostate cancer cells Gamma-tocotrienol suppresses prostate cancer cell proliferation and invasion through multiple-signalling pathways Evidence of gamma-tocotrienol as an apoptosis-inducing, invasion-suppressing, and chemotherapy drug-sensitizing agent in human melanoma cells Exit from arsenite-induced mitotic arrest is p53 dependent Inhibitor of differentiation 1 promotes endothelial survival in a bleomycin model of lung injury in mice Bone morphogenetic protein 4, inhibitor of differentiation 1, and epidermal growth factor receptor regulate the survival of cochlear sensory epithelial cells Cancer biomarker discovery: the entropic hallmark.Inhibition of Id proteins by a peptide aptamer induces cell-cycle arrest and apoptosis in ovarian cancer cells.ID2 predicts poor prognosis in breast cancer, especially in triple-negative breast cancer, and inhibits E-cadherin expression Chemopreventive effect of PSP through targeting of prostate cancer stem cell-like population.LIF negatively regulates tumour-suppressor p53 through Stat3/ID1/MDM2 in colorectal cancers.High throughput RNAi screening identifies ID1 as a synthetic sick/lethal gene interacting with the common TP53 mutation R175H.ID proteins regulate diverse aspects of cancer progression and provide novel therapeutic opportunities.Insulin-like growth factor I suppresses bone morphogenetic protein signaling in prostate cancer cells by activating mTOR signaling Adaptive response of vascular endothelial cells to an acute increase in shear stress magnitude Tubular cell dedifferentiation and peritubular inflammation are coupled by the transcription regulator Id1 in renal fibrogenesis.Physical and functional interaction between the ID1 and p65 for activation of NF-κB Nuclear localisation of nuclear factor-kappaB transcription factors in prostate cancer: an immunohistochemical study.Id-I stimulates cell proliferation through activation of EGFR in ovarian cancer cells Id1 is a common downstream target of oncogenic tyrosine kinases in leukemic cells Expression of the inhibitor of DNA-binding (ID)-1 protein as an angiogenic mediator in tumour advancement of uterine cervical cancers.Hexosamine template. A platform for modulating gene expression and for sugar-based drug discovery Regulation of Id1 expression by epigallocatechin‑3‑gallate and its effect on the proliferation and apoptosis of poorly differentiated AGS gastric cancer cells.Cinchonine induces apoptosis of HeLa and A549 cells through targeting TRAF6.Id-1 gene downregulation by sulindac sulfide and its upregulation during tumor development in gastric cancer.Apigenin inhibits proliferation of ovarian cancer A2780 cells through Id1.Id1 overexpression induces tetraploidization and multiple abnormal mitotic phenotypes by modulating aurora A Footprintless disruption of prosurvival genes in aneuploid cancer cells using CRISPR/Cas9 technology.Effects of upregulation of Id3 in human lung adenocarcinoma cells on proliferation, apoptosis, mobility and tumorigenicity.Id1 and NF-κB promote the generation of CD133+ and BMI-1+ keratinocytes and the growth of xenograft tumors in mice.Id4 promotes senescence and sensitivity to doxorubicin-induced apoptosis in DU145 prostate cancer cells.The Id-protein family in developmental and cancer-associated pathways.Daxx regulates mitotic progression and prostate cancer predisposition.Effect of Id1 knockdown on formation of osteolytic bone lesions by prostate cancer PC3 cells in vivo.Expression of Id proteins is regulated by the Bcl-3 proto-oncogene in prostate cancer.Id-1 and the p65 subunit of NF-κB promote migration of nasopharyngeal carcinoma cells and are correlated with poor prognosis.ID4 is frequently downregulated and partially hypermethylated in prostate cancer.Using self-assembled nanomaterials to inhibit the formation of metastatic cancer stem cell colonies in vitro.Gamma-tocotrienol as an effective agent in targeting prostate cancer stem cell-like population.ID1 enhances docetaxel cytotoxicity in prostate cancer cells through inhibition of p21.

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Id-1 expression promotes cell survival through activation of NF-kappaB signalling pathway in prostate cancer cells.

http://www.wikidata.org/entity/Q38352246

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Id-1 expression promotes cell ...... hway in prostate cancer cells.

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Id-1 expression promotes cell ...... hway in prostate cancer cells.

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Kexin Xu

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Ming-Tat Ling

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Sai-Wah Tsao

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Xianghong Wang

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Xue-Song Ouyang

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Yong-Chuan Wong

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P2860

The NF-kappa B and I kappa B proteins: new discoveries and insights

IEX-1L, an apoptosis inhibitor involved in NF-kappaB-mediated cell survival

Mapping of the inducible IkappaB phosphorylation sites that signal its ubiquitination and degradation

New insights into the role of nuclear factor-kB in cell growth regulation

Id1 and Id3 are required for neurogenesis, angiogenesis and vascularization of tumour xenografts

Opposing effects of Ets and Id proteins on p16INK4a expression during cellular senescence

Id-1 stimulates serum independent prostate cancer cell proliferation through inactivation of p16(INK4a)/pRB pathway

Down-regulation of Id-1 expression is associated with TGF beta 1-induced growth arrest in prostate epithelial cells

The protein Id: a negative regulator of helix-loop-helix DNA binding proteins

Biochemical characteristics of caspases-3, -6, -7, and -8

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