Identification of cJun-responsive genes in Rat-1a cells using multiple techniques: increased expression of stathmin is necessary for cJun-mediated anchorage-independent growth.
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Clinicopathological significance of expression of p-c-Jun, TCF4 and beta-Catenin in colorectal tumorsp21-activated kinase 3 (PAK3) is an AP-1 regulated gene contributing to actin organisation and migration of transformed fibroblastsCell fate determination factor DACH1 inhibits c-Jun-induced contact-independent growthCyclin A is a c-Jun target gene and is necessary for c-Jun-induced anchorage-independent growth in RAT1a cellsp75-Ras-GRF1 is a c-Jun/AP-1 target protein: its up regulation results in increased Ras activity and is necessary for c-Jun-induced nonadherent growth of Rat1a cellsDisruption of c-Jun reduces cellular migration and invasion through inhibition of c-Src and hyperactivation of ROCK II kinase.Emerging roles of ATF2 and the dynamic AP1 network in cancer.Mammary gland selective excision of c-jun identifies its role in mRNA splicing.Stathmin regulates keratinocyte proliferation and migration during cutaneous regeneration.Inducible overexpression of c-Jun in MCF7 cells causes resistance to vinblastine via inhibition of drug-induced apoptosis and senescence at a step subsequent to mitotic arrest.Genetic analysis of genes causing hypertension and stroke in spontaneously hypertensive rats: Gene expression profiles in the kidneys.Growth inhibition of non-small cell lung cancer cells by AP-1 blockade using a cJun dominant-negative mutant.A novel role for jun N-terminal kinase signaling in olfactory sensory neuronal death.Simultaneous blockade of AP-1 and phosphatidylinositol 3-kinase pathway in non-small cell lung cancer cellsInhibition of AP-1 transcriptional activity blocks the migration, invasion, and experimental metastasis of murine osteosarcoma.RSK2 signals through stathmin to promote microtubule dynamics and tumor metastasisStathmin: a protein with many tasks. New biomarker and potential target in cancer.c-Jun enhances intestinal epithelial restitution after wounding by increasing phospholipase C-γ1 transcription.Inhibition of AP-1 suppresses cervical cancer cell proliferation and is associated with p21 expression.PMA-induced up-regulation of TBX3 is mediated by AP-1 and contributes to breast cancer cell migration.Altered regulation of c-jun and its involvement in anchorage-independent growth of human lung cancers.Transcriptional tools: Small molecules for modulating CBP KIX-dependent transcriptional activators.c-Jun N-terminal kinase/c-Jun inhibits fibroblast proliferation by negatively regulating the levels of stathmin/oncoprotein 18.
P2860
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P2860
Identification of cJun-responsive genes in Rat-1a cells using multiple techniques: increased expression of stathmin is necessary for cJun-mediated anchorage-independent growth.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh
2003年學術文章
@zh-hant
name
Identification of cJun-respons ...... anchorage-independent growth.
@en
type
label
Identification of cJun-respons ...... anchorage-independent growth.
@en
prefLabel
Identification of cJun-respons ...... anchorage-independent growth.
@en
P2093
P2860
P356
P1433
P1476
Identification of cJun-respons ...... anchorage-independent growth.
@en
P2093
Anita Sabichi
Ichiro Kinoshita
Michael J Birrer
Motoo Katabami
Ramon G Manzano
Virna Leaner
P2860
P2888
P304
P356
10.1038/SJ.ONC.1206371
P407
P50
P577
2003-05-01T00:00:00Z
P5875
P6179
1007699182