The dual effect of adenovirus type 5 E1A 13S protein on NF-kappaB activation is antagonized by E1B 19K.
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INK4 cell cycle inhibitors direct transcriptional inactivation of NF-kappaBBtf, a novel death-promoting transcriptional repressor that interacts with Bcl-2-related proteins.Expression of hepatitis C virus proteins inhibits signal transduction through the Jak-STAT pathwayGlucocorticoid-mediated repression of nuclear factor-kappaB-dependent transcription involves direct interference with transactivationImpact of E1 and Cre on adenovirus vector amplification: developing MDCK CAV-2-E1 and E1-Cre transcomplementing cell lines.The oncolytic virus dl922-947 reduces IL-8/CXCL8 and MCP-1/CCL2 expression and impairs angiogenesis and macrophage infiltration in anaplastic thyroid carcinoma.The adenovirus e3 promoter is sensitive to activation signals in human T cells.Adenovirus type 5 rupture of lysosomes leads to cathepsin B-dependent mitochondrial stress and production of reactive oxygen species.Promyelocytic Leukemia Protein Isoform II Promotes Transcription Factor Recruitment To Activate Interferon Beta and Interferon-Responsive Gene Expression.Basal transcription factors TBP and TFIIB and the viral coactivator E1A 13S bind with distinct affinities and kinetics to the transactivation domain of NF-kappaB p65.Promyelocytic leukemia protein isoform II inhibits infection by human adenovirus type 5 through effects on HSP70 and the interferon response.Identification of a novel antiapoptotic protein, GAM-1, encoded by the CELO adenovirus.Contributions of E1A to mouse adenovirus type 1 pathogenesis following intranasal inoculation.Functional interactions of antiapoptotic proteins and tumor necrosis factor in the context of a replication-competent adenovirus.Chromatin-dependent E1A activity modulates NF-kappaB RelA-mediated repression of glucocorticoid receptor-dependent transcription.The human papillomavirus oncoprotein E7 attenuates NF-kappa B activation by targeting the Ikappa B kinase complex.Tumor necrosis factor-alpha-induced cell killing and activation of transcription factor NF-kappaB are uncoupled in L929 cells.p38 and extracellular signal-regulated kinase mitogen-activated protein kinase pathways are required for nuclear factor-kappaB p65 transactivation mediated by tumor necrosis factor.Distinct domains of the RelA NF-kappaB subunit are required for negative cross-talk and direct interaction with the glucocorticoid receptor.T cell receptor-induced lipid raft recruitment of the I kappa B kinase complex is necessary and sufficient for NF-kappa B activation occurring in the cytosol.Zinc finger and carboxyl regions of adenovirus E1A 13S CR3 are important for transactivation of the cytomegalovirus major immediate early promoter by adenovirus.NF-κB-mediated Induction ofmdr1bExpression by Insulin in Rat Hepatoma Cells
P2860
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P2860
The dual effect of adenovirus type 5 E1A 13S protein on NF-kappaB activation is antagonized by E1B 19K.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
The dual effect of adenovirus ...... ion is antagonized by E1B 19K.
@en
type
label
The dual effect of adenovirus ...... ion is antagonized by E1B 19K.
@en
prefLabel
The dual effect of adenovirus ...... ion is antagonized by E1B 19K.
@en
P2093
P2860
P356
P1476
The dual effect of adenovirus ...... ion is antagonized by E1B 19K.
@en
P2093
Baeuerle PA
Limbourg FP
Schmitz ML
Städtler H
Traenckner EB
P2860
P304
P356
10.1128/MCB.16.8.4052
P407
P577
1996-08-01T00:00:00Z