Slug, a highly conserved zinc finger transcriptional repressor, protects hematopoietic progenitor cells from radiation-induced apoptosis in vivo.
about
Negative regulation of the expressions of cytokeratins 8 and 19 by SLUG repressor protein in human breast cellsAberrant expression of the transcription factors snail and slug alters the response to genotoxic stressSlug is a direct Notch target required for initiation of cardiac cushion cellularizationNew Insights into the Crossroads between EMT and Stemness in the Context of CancerEpithelial-mesenchymal plasticity in carcinoma metastasisInduction of the neural crest state: control of stem cell attributes by gene regulatory, post-transcriptional and epigenetic interactionsSnail transcription factors in hematopoietic cell development: a model of functional redundancyAdipose tissue mass is modulated by SLUG (SNAI2)Slug deficiency enhances self-renewal of hematopoietic stem cells during hematopoietic regenerationFunctional study of transcription factor KLF11 by targeted gene inactivationSnail blocks the cell cycle and confers resistance to cell deathMembrane-anchored uPAR regulates the proliferation, marrow pool size, engraftment, and mobilization of mouse hematopoietic stem/progenitor cells.Epiblast-specific Snai1 deletion results in embryonic lethality due to multiple vascular defects.An NF-kappaB and slug regulatory loop active in early vertebrate mesoderm.Potential Involvement of Snail Members in Neuronal Survival and Astrocytic Migration during the Gecko Spinal Cord RegenerationSLUG/SNAI2 and tumor necrosis factor generate breast cells with CD44+/CD24- phenotype.Snail: More than EMTThe Snail transcription factor regulates the numbers of neural precursor cells and newborn neurons throughout mammalian lifeCore epithelial-to-mesenchymal transition interactome gene-expression signature is associated with claudin-low and metaplastic breast cancer subtypesZebrafish arl6ip1 is required for neural crest development during embryogenesisThe epithelial-mesenchymal transition (EMT) regulatory factor SLUG (SNAI2) is a downstream target of SPARC and AKT in promoting melanoma cell invasionSFRP2 and slug contribute to cellular resistance to apoptosis in hypertrophic scarsHoxa-10 deficiency alters region-specific gene expression and perturbs differentiation of natural killer cells during decidualizationDeletion of Snai2 and Snai3 results in impaired physical development compounded by lymphocyte deficiency.Aberrant expression of osteopontin and E-cadherin indicates radiation resistance and poor prognosis for patients with cervical carcinomaMiR-124 Radiosensitizes human colorectal cancer cells by targeting PRRX1Loss of Snail2 favors skin tumor progression by promoting the recruitment of myeloid progenitors.Screening of suppressors of bax-induced cell death identifies glycerophosphate oxidase-1 as a mediator of debcl-induced apoptosis in Drosophila.Slug is required for cell survival during partial epithelial-mesenchymal transition of HGF-induced tubulogenesisAnterior gradient protein 2 expression in high grade head and neck squamous cell carcinoma correlated with cancer stem cell and epithelial mesenchymal transition.Roles of the transcription factors snail and slug during mammary morphogenesis and breast carcinoma progression.Regulation of the protein stability of EMT transcription factorsGenomics and drug profiling of fatal TCF3-HLF-positive acute lymphoblastic leukemia identifies recurrent mutation patterns and therapeutic options.Inhibition of Snail Family Transcriptional Repressor 2 (SNAI2) Enhances Multidrug Resistance of Hepatocellular Carcinoma CellsGene therapy for radioprotection.SLUG is a direct transcriptional repressor of PTEN tumor suppressor.Gene and protein analysis reveals that p53 pathway is functionally inactivated in cytogenetically normal Acute Myeloid Leukemia and Acute Promyelocytic LeukemiaClues from worms: a Slug at Puma promotes the survival of blood progenitors.Basal/HER2 breast carcinomas: integrating molecular taxonomy with cancer stem cell dynamics to predict primary resistance to trastuzumab (Herceptin).Snai2 and Snai3 transcriptionally regulate cellular fitness and functionality of T cell lineages through distinct gene programs.
P2860
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P2860
Slug, a highly conserved zinc finger transcriptional repressor, protects hematopoietic progenitor cells from radiation-induced apoptosis in vivo.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
Slug, a highly conserved zinc ...... ion-induced apoptosis in vivo.
@en
type
label
Slug, a highly conserved zinc ...... ion-induced apoptosis in vivo.
@en
prefLabel
Slug, a highly conserved zinc ...... ion-induced apoptosis in vivo.
@en
P2093
P1433
P1476
Slug, a highly conserved zinc ...... tion-induced apoptosis in vivo
@en
P2093
A Thomas Look
Adolfo A Ferrando
Akira Inoue
Akira Morimoto
Carl W Jackson
Gerard C Grosveld
Hiromi Iwasaki
Johann K Hitzler
Koichi Akashi
Miriam J Chong
P304
P356
10.1016/S1535-6108(02)00155-1
P577
2002-10-01T00:00:00Z