Inflammation in cardiac injury, repair and regeneration.
about
The emerging role of Toll-like receptor 4 in myocardial inflammationModulators of Macrophage Polarization Influence Healing of the Infarcted MyocardiumWild-type p53-induced Phosphatase 1 Deficiency Exacerbates Myocardial Infarction-induced Ischemic Injury.The role of Interleukin Receptor Associated Kinase (IRAK)-M in regulation of myofibroblast phenotype in vitro, and in an experimental model of non-reperfused myocardial infarction.Metabolically-inactive glucagon-like peptide-1(9-36)amide confers selective protective actions against post-myocardial infarction remodelling.Resolution of Acute Inflammation and the Role of Resolvins in Immunity, Thrombosis, and Vascular Biology.Inflammation as a therapeutic target in myocardial infarction: learning from past failures to meet future challenges.Neuregulin-1β induces proliferation, survival and paracrine signaling in normal human cardiac ventricular fibroblasts.The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis.How cardiomyocytes sense pathophysiological stresses for cardiac remodeling.GLP-1 receptor independent pathways: emerging beneficial effects of GLP-1 breakdown products.Ischemia/Reperfusion.Monocytes increase human cardiac myofibroblast-mediated extracellular matrix remodeling through TGF-β1.Loss of Macrophage Wnt Secretion Improves Remodeling and Function After Myocardial Infarction in Mice.Diabetes and Cardioplegia.Myocardial Reparative Properties of Cardiac Mesenchymal Cells Isolated on the Basis of AdherenceIL-10 improves cardiac remodeling after myocardial infarction by stimulating M2 macrophage polarization and fibroblast activation.FoxO4 promotes early inflammatory response upon myocardial infarction via endothelial Arg1.Elevated plasma levels of interleukin-16 in patients with acute myocardial infarction.Sanggenon C protects against cardiomyocyte hypoxia injury by increasing autophagy.Identification of potentially relevant genes for myocardial infarction using RNA sequencing data analysis.Guidelines for Experimental Models of Myocardial Ischemia and Infarction.Leptin-induced inflammation by activating IL-6 expression contributes to the fibrosis and hypertrophy of ligamentum flavum in lumbar spinal canal stenosis.Molecular mechanisms of cardioprotective effects mediated by transplanted cardiac ckit+ cells through the activation of an inflammatory hypoxia-dependent reparative response.Alcohol attenuates myocardial ischemic injury.Lymphocytes at the Heart of Wound Healing.Endotoxin, Toll-like Receptor-4, and Atherosclerotic Heart Disease.Beta-catenin promotes macrophage-mediated acute inflammatory response after myocardial infarction.IL-18 cleavage triggers cardiac inflammation and fibrosis upon β-adrenergic insult.Endotoxin, Toll-like Receptor-4, and Atherosclerotic Heart Disease.Annexin A5 reduces infarct size and improves cardiac function after myocardial ischemia-reperfusion injury by suppression of the cardiac inflammatory response.Apoptosis and Mobilization of Lymphocytes to Cardiac Tissue Is Associated with Myocardial Infarction in a Reperfused Porcine Model and Infarct Size in Post-PCI Patients.Role of TGF‑β1 expressed in bone marrow‑derived mesenchymal stem cells in promoting bone formation in a rabbit femoral defect model.
P2860
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P2860
Inflammation in cardiac injury, repair and regeneration.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Inflammation in cardiac injury, repair and regeneration.
@en
type
label
Inflammation in cardiac injury, repair and regeneration.
@en
prefLabel
Inflammation in cardiac injury, repair and regeneration.
@en
P2860
P1476
Inflammation in cardiac injury, repair and regeneration
@en
P2093
Nikolaos G Frangogiannis
P2860
P304
P356
10.1097/HCO.0000000000000158
P577
2015-05-01T00:00:00Z