Glomerular expression of dystroglycans is reduced in minimal change nephrosis but not in focal segmental glomerulosclerosis. - Wikidata - awgldk - TriplyDB

Glomerular expression of dystroglycans is reduced in minimal change nephrosis but not in focal segmental glomerulosclerosis.

Glomerular expression of dystroglycans is reduced in minimal change nephrosis but not in focal segmental glomerulosclerosis.

http://www.wikidata.org/entity/Q38465690

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Glomerular podocytes possess the synaptic vesicle molecule Rab3A and its specific effector rabphilin-3a Podocytes Cell biology and pathology of podocytes Novel siRNA delivery system to target podocytes in vivo Podocyte biology and pathogenesis of kidney disease Ligation of alpha-dystroglycan on podocytes induces intracellular signaling: a new mechanism for podocyte effacement?Corticosteroids vs. corticosteroids plus cycloporin A in adult minimal changes disease alpha- and beta-Adducin polymorphisms affect podocyte proteins and proteinuria in rodents and decline of renal function in human IgA nephropathy.Induction of B7-1 in podocytes is associated with nephrotic syndrome Proteinuria: an enzymatic disease of the podocyte?Lack of collagen XVIII long isoforms affects kidney podocytes, whereas the short form is needed in the proximal tubular basement membrane.Dystroglycan in the diagnosis of FSGS.Dystroglycan does not contribute significantly to kidney development or function, in health or after injury.The LIM-homeodomain transcription factor Lmx1b plays a crucial role in podocytes.Glomerular proteinuria: a complex interplay between unique players.Proteolytic processing of dynamin by cytoplasmic cathepsin L is a mechanism for proteinuric kidney disease.Conformational transitions and glycation of serum albumin in patients with minimal-change glomerulopathy.HANAC Syndrome Col4a1 Mutation Causes Neonate Glomerular Hyperpermeability and Adult Glomerulocystic Kidney Disease.Novel biomarkers in glomerular disease.Podocin and beta dystroglycan expression to study podocyte-podocyte and basement membrane matrix connections in adult protienuric states.Role of mindin in diabetic nephropathy.The role of cell-extracellular matrix interactions in glomerular injury.Pathophysiology of the diabetic kidney.Renal biopsy: use of biomarkers as a tool for the diagnosis of focal segmental glomerulosclerosis.Causes and pathogenesis of focal segmental glomerulosclerosis.Molecular and genetic basis of inherited nephrotic syndrome Human genetics of nephrotic syndrome and the quest for precision medicine.Caught flat-footed: podocyte damage and the molecular bases of focal glomerulosclerosis.The reno-protective effect of a phosphoinositide 3-kinase inhibitor wortmannin on streptozotocin-induced proteinuric renal disease rats.Glycosaminoglycan regulation by VEGFA and VEGFC of the glomerular microvascular endothelial cell glycocalyx in vitro.Detection of activated parietal epithelial cells on the glomerular tuft distinguishes early focal segmental glomerulosclerosis from minimal change disease.Prospects for Precision Medicine in Glomerulonephritis Treatment.

P2860

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P2860

Glomerular expression of dystroglycans is reduced in minimal change nephrosis but not in focal segmental glomerulosclerosis.

http://www.wikidata.org/entity/Q38465690

description

2000 nî lūn-bûn

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2000年の論文

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2000年論文

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2000年論文

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2000年論文

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2000年論文

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2000年論文

@zh-tw

2000年论文

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2000年论文

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2000年论文

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name

Glomerular expression of dystr ...... segmental glomerulosclerosis.

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type

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Glomerular expression of dystr ...... segmental glomerulosclerosis.

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prefLabel

Glomerular expression of dystr ...... segmental glomerulosclerosis.

@en

P1433

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Journal of the American Society of Nephrology

P1476

Glomerular expression of dystr ...... l segmental glomerulosclerosis

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P2093

Bittner RE

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Fillipovic E

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Kerjaschki D

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Kraxberger I

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Langer B

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Poczewki H

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Regele HM

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403-412

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3

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2000-03-01T00:00:00Z

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