about
Global gene expression analysis identifies PDEF transcriptional networks regulating cell migration during cancer progressionA glutathione S-transferase pi-activated prodrug causes kinase activation concurrent with S-glutathionylation of proteins.Epithelial-to-mesenchymal transition and the cancer stem cell phenotype: insights from cancer biology with therapeutic implications for colorectal cancer.Distinct regulatory proteins control the graded transcriptional response to increasing H(2)O(2) levels in fission yeast Schizosaccharomyces pombe.AGE metabolites: a biomarker linked to cancer disparity?Staphylococcus epidermidis recovered from indwelling catheters exhibit enhanced biofilm dispersal and "self-renewal" through downregulation of agrFLI1 expression is correlated with breast cancer cellular growth, migration, and invasion and altered gene expression.SNAI2 modulates colorectal cancer 5-fluorouracil sensitivity through miR145 repressionA novel role for human sulfiredoxin in the reversal of glutathionylation.Prostate-Derived ETS Factor Regulates Epithelial-to-Mesenchymal Transition through Both SLUG-Dependent and Independent MechanismsMechanisms and functional consequences of PDEF protein expression loss during prostate cancer progression.Slug expression enhances tumor formation in a noninvasive rectal cancer model.Hematopoietic stem cell-derived cancer-associated fibroblasts are novel contributors to the pro-tumorigenic microenvironmentKSHV-Encoded MicroRNAs: Lessons for Viral Cancer Pathogenesis and Emerging ConceptsSulfiredoxin: a potential therapeutic agent?Glutathione S-transferases as regulators of kinase pathways and anticancer drug targets.Kaposi's sarcoma-associated herpesvirus suppression of DUSP1 facilitates cellular pathogenesis following de novo infection.Defining ETS transcription regulatory networks and their contribution to breast cancer progression.MicroRNA-mediated inhibition of prostate-derived Ets factor messenger RNA translation affects prostate-derived Ets factor regulatory networks in human breast cancer.MicroRNA-510 promotes cell and tumor growth by targeting peroxiredoxin1 in breast cancer.MicroRNAs and Their Impact on Breast Cancer, the Tumor Microenvironment, and Disparities.ETS1 transcriptional activity is increased in advanced prostate cancer and promotes the castrate-resistant phenotype.PABA/NO as an anticancer lead: analogue synthesis, structure revision, solution chemistry, reactivity toward glutathione, and in vitro activity.Tumor cell responses to a novel glutathione S-transferase-activated nitric oxide-releasing prodrug.Slug expression inhibits calcitriol-mediated sensitivity to radiation in colorectal cancer.A 2-Cys peroxiredoxin regulates peroxide-induced oxidation and activation of a stress-activated MAP kinase.Oxidation of a eukaryotic 2-Cys peroxiredoxin is a molecular switch controlling the transcriptional response to increasing levels of hydrogen peroxide.
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description
hulumtuese
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Victoria J. Findlay
@ast
Victoria J. Findlay
@en
Victoria J. Findlay
@es
Victoria J. Findlay
@sl
type
label
Victoria J. Findlay
@ast
Victoria J. Findlay
@en
Victoria J. Findlay
@es
Victoria J. Findlay
@sl
altLabel
Findlay VJ
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prefLabel
Victoria J. Findlay
@ast
Victoria J. Findlay
@en
Victoria J. Findlay
@es
Victoria J. Findlay
@sl
P106
P1153
6602597280
P21
P31
P496
0000-0002-9994-6319