Mitochondrial Ca(2+) Remodeling is a Prime Factor in Oncogenic Behavior.
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The Concerted Action of Mitochondrial Dynamics and Positioning: New Characters in Cancer Onset and Progression.Mcl-1 involvement in mitochondrial dynamics is associated with apoptotic cell deathGlycolysis and oxidative phosphorylation are essential for purinergic receptor-mediated angiogenic responses in vasa vasorum endothelial cells.The Mitochondrial Voltage-Dependent Anion Channel 1, Ca2+ Transport, Apoptosis, and Their Regulation.Intersection of mitochondrial fission and fusion machinery with apoptotic pathways: Role of Mcl-1.Orchestrating cellular signaling pathways-the cellular "conductor" protein tyrosine phosphatase interacting protein 51 (PTPIP51).Coiled-coil domain containing 109B is a HIF1α-regulated gene critical for progression of human gliomas.The Influence of MicroRNAs on Mitochondrial CalciumThe Cooperative Relationship between STAT5 and Reactive Oxygen Species in Leukemia: Mechanism and Therapeutic Potential
P2860
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P2860
Mitochondrial Ca(2+) Remodeling is a Prime Factor in Oncogenic Behavior.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
Mitochondrial Ca(2+) Remodeling is a Prime Factor in Oncogenic Behavior.
@en
type
label
Mitochondrial Ca(2+) Remodeling is a Prime Factor in Oncogenic Behavior.
@en
prefLabel
Mitochondrial Ca(2+) Remodeling is a Prime Factor in Oncogenic Behavior.
@en
P2860
P50
P356
P1476
Mitochondrial Ca(2+) Remodeling is a Prime Factor in Oncogenic Behavior.
@en
P2093
Massimo Bonora
Simone Patergnani
P2860
P356
10.3389/FONC.2015.00143
P577
2015-06-25T00:00:00Z