Elongation factor 2 kinase promotes cell survival by inhibiting protein synthesis without inducing autophagy.
about
Decreased ATF4 expression as a mechanism of acquired resistance to long-term amino acid limitation in cancer cellsAcute lymphoblastic leukemia cells are sensitive to disturbances in protein homeostasis induced by proteasome deubiquitinase inhibitionEukaryotic elongation factor 2 kinase upregulates the expression of proteins implicated in cell migration and cancer cell metastasis.Cross-talk between protein synthesis, energy metabolism and autophagy in cancer.Intrauterine growth restriction inhibits expression of eukaryotic elongation factor 2 kinase, a regulator of protein translation.
P2860
Elongation factor 2 kinase promotes cell survival by inhibiting protein synthesis without inducing autophagy.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Elongation factor 2 kinase pro ...... is without inducing autophagy.
@en
type
label
Elongation factor 2 kinase pro ...... is without inducing autophagy.
@en
prefLabel
Elongation factor 2 kinase pro ...... is without inducing autophagy.
@en
P2093
P2860
P50
P1433
P1476
Elongation factor 2 kinase pro ...... is without inducing autophagy.
@en
P2093
Claire E J Moore
Jo Pickford
John Barron
Matthias Versele
Xuemin Wang
P2860
P304
P356
10.1016/J.CELLSIG.2016.01.005
P577
2016-01-18T00:00:00Z