LXRα regulates macrophage arginase 1 through PU.1 and interferon regulatory factor 8.
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Selective endothelial overexpression of arginase II induces endothelial dysfunction and hypertension and enhances atherosclerosis in miceLiver X Receptors Link Lipid Metabolism and Inflammation.Phenotypic polarization of macrophages in atherosclerosisArginase 2 deficiency prevents oxidative stress and limits hyperoxia-induced retinal vascular degeneration.Foam Cell Formation In Vivo Converts Macrophages to a Pro-Fibrotic PhenotypeNuclear Receptor Nr4a2 Promotes Alternative Polarization of Macrophages and Confers Protection in Sepsis.New Lives Given by Cell Death: Macrophage Differentiation Following Their Encounter with Apoptotic Leukocytes during the Resolution of InflammationRegulation of reverse cholesterol transport - a comprehensive appraisal of available animal studies.Macrophages, dendritic cells, and regression of atherosclerosis.Gene Therapy Targeting LDL Cholesterol but not HDL Cholesterol Induces Regression of Advanced Atherosclerosis in a Mouse Model of Familial HypercholesterolemiaRBP-J is required for M2 macrophage polarization in response to chitin and mediates expression of a subset of M2 genesThe nuclear receptor LXR modulates interleukin-18 levels in macrophages through multiple mechanisms.Nuclear receptors in inflammation control: repression by GR and beyond.Polarization of M2 macrophages requires Lamtor1 that integrates cytokine and amino-acid signals.Interferon regulatory factor 8 modulates phenotypic switching of smooth muscle cells by regulating the activity of myocardin.Molecular pathways regulating macrophage polarization: implications for atherosclerosis.Liver X receptors, atherosclerosis and inflammation.Biological roles of liver X receptors in immune cells.Hanging in the balance: endogenous anti-inflammatory mechanisms in tissue repair and fibrosis.Molecular biology of atherosclerosis.Modulation of Macrophage Gene Expression via Liver X Receptor α Serine 198 Phosphorylation.The Intracellular Cholesterol Landscape: Dynamic Integrator of the Immune Response.The challenges and promise of targeting the Liver X Receptors for treatment of inflammatory disease.Minimal PU.1 reduction induces a preleukemic state and promotes development of acute myeloid leukemia.Retinoic acid promotes the development of Arg1-expressing dendritic cells for the regulation of T-cell differentiation.Rev-erb-α regulates atrophy-related genes to control skeletal muscle mass.Modulation of chitotriosidase during macrophage differentiation.Mer signaling increases the abundance of the transcription factor LXR to promote the resolution of acute sterile inflammation.Beyond the Foam Cell: The Role of LXRs in Preventing Atherogenesis
P2860
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P2860
LXRα regulates macrophage arginase 1 through PU.1 and interferon regulatory factor 8.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
LXRα regulates macrophage arginase 1 through PU.1 and interferon regulatory factor 8.
@en
type
label
LXRα regulates macrophage arginase 1 through PU.1 and interferon regulatory factor 8.
@en
prefLabel
LXRα regulates macrophage arginase 1 through PU.1 and interferon regulatory factor 8.
@en
P2093
P2860
P50
P1433
P1476
LXRα regulates macrophage arginase 1 through PU.1 and interferon regulatory factor 8
@en
P2093
Diane Kepka-Lenhart
Jonathan E Feig
Michael J Garabedian
Sidney M Morris
Yuliya Vengrenyuk
P2860
P304
P356
10.1161/CIRCRESAHA.111.241810
P577
2011-07-14T00:00:00Z