Mitochondrial dysfunction and seizures: the neuronal energy crisis.
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Direct Imaging of Hippocampal Epileptiform Calcium Motifs Following Kainic Acid Administration in Freely Behaving MiceNeuronal Hyperactivity Disturbs ATP Microgradients, Impairs Microglial Motility, and Reduces Phagocytic Receptor Expression Triggering Apoptosis/Microglial Phagocytosis UncouplingBGP-15 Protects against Oxidative Stress- or Lipopolysaccharide-Induced Mitochondrial Destabilization and Reduces Mitochondrial Production of Reactive Oxygen SpeciesMutations in the Na(+)/citrate cotransporter NaCT (SLC13A5) in pediatric patients with epilepsy and developmental delayMyoinositol Attenuates the Cell Loss and Biochemical Changes Induced by Kainic Acid Status Epilepticus.Epilepsy due to mutations in the mitochondrial polymerase gamma (POLG) gene: A clinical and molecular genetic review.Glucose Deficiency Elevates Acid-Sensing Ion Channel 2a Expression and Increases Seizure Susceptibility in Temporal Lobe EpilepsyNeurological disorders associated with DNA strand-break processing enzymes.Chromosomal microarray testing identifies a 4p terminal region associated with seizures in Wolf-Hirschhorn syndrome.SIRT5 Deficiency Enhances Susceptibility to Kainate-Induced Seizures and Exacerbates Hippocampal Neurodegeneration not through Mitochondrial Antioxidant Enzyme SOD2.Migraine in mitochondrial disorders: Prevalence and characteristics.Post-epilepsy stroke: A review.In vitro acute and developmental neurotoxicity screening: an overview of cellular platforms and high-throughput technical possibilities.The vicious circle of hypometabolism in neurodegenerative diseases: Ways and mechanisms of metabolic correction.Mechanisms of Excessive Extracellular Glutamate Accumulation in Temporal Lobe Epilepsy.Differential effects of energy deprivation on intracellular sodium homeostasis in neurons and astrocytes.Plasma Membrane Na⁺-Coupled Citrate Transporter (SLC13A5) and Neonatal Epileptic Encephalopathy.Effect of hyperbaric oxygenation on mitochondrial function of neuronal cells in the cortex of neonatal rats after hypoxic-ischemic brain damage.Cancer: Linking Powerhouses to Suicidal Bags.Metabolic and Homeostatic Changes in Seizures and Acquired Epilepsy-Mitochondria, Calcium Dynamics and Reactive Oxygen Species.Mitochondrial Liver Toxicity of Valproic Acid and Its Acid Derivatives Is Related to Inhibition of α-Lipoamide Dehydrogenase.Contribution of Intrinsic Lactate to Maintenance of Seizure Activity in Neocortical Slices from Patients with Temporal Lobe Epilepsy and in Rat Entorhinal Cortex.NMNAT3 is protective against the effects of neonatal cerebral hypoxia-ischemia.Neuropathological signs of inflammation correlate with mitochondrial DNA deletions in mesial temporal lobe epilepsy.Increase in Mitochondrial Biogenesis in Neuronal Cells by RNS60, a Physically-Modified Saline, via Phosphatidylinositol 3-Kinase-Mediated Upregulation of PGC1α.The novel anticonvulsant neuropeptide and galanin analogue, NAX-5055, does not alter energy and amino acid metabolism in cultured brain cells.Extracellular HMGB1 Modulates Glutamate Metabolism Associated with Kainic Acid-Induced Epilepsy-Like Hyperactivity in Primary Rat Neural Cells.Near infrared spectroscopy with a vascular occlusion test as a biomarker in children with mitochondrial and other neuro-genetic disordersBioenergetic Mechanisms of Seizure Control
P2860
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P2860
Mitochondrial dysfunction and seizures: the neuronal energy crisis.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Mitochondrial dysfunction and seizures: the neuronal energy crisis.
@en
type
label
Mitochondrial dysfunction and seizures: the neuronal energy crisis.
@en
prefLabel
Mitochondrial dysfunction and seizures: the neuronal energy crisis.
@en
P1433
P1476
Mitochondrial dysfunction and seizures: the neuronal energy crisis.
@en
P304
P356
10.1016/S1474-4422(15)00148-9
P577
2015-09-01T00:00:00Z