Mechanical overload induces VEGF in cartilage discs via hypoxia-inducible factor. - Wikidata - awgldk - TriplyDB

Mechanical overload induces VEGF in cartilage discs via hypoxia-inducible factor.

Mechanical overload induces VEGF in cartilage discs via hypoxia-inducible factor.

http://www.wikidata.org/entity/Q38584496

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Oxidation in rheumatoid arthritis.Regulation of type II collagen synthesis during osteoarthritis by prolyl-4-hydroxylases: possible influence of low oxygen levels Animal models of osteoarthritis: classification, update, and measurement of outcomes Angiopoietin-like 4 promotes angiogenesis in the tendon and is increased in cyclically loaded tendon fibroblasts 17β-estradiol reduces expression of MMP-1, -3, and -13 in human primary articular chondrocytes from female patients cultured in a three dimensional alginate system.The association between retraction of the torn rotator cuff and increasing expression of hypoxia inducible factor 1α and vascular endothelial growth factor expression: an immunohistological study.Signaling pathways in cartilage repair.Emerging roles of SUMO modification in arthritis.Joint pathology and platelet-rich plasma therapies.Intravenous administration of anti-vascular endothelial growth factor humanized monoclonal antibody bevacizumab improves articular cartilage repair Role of the endothelium in inflammatory bowel diseases.Blocking vascular endothelial growth factor with soluble Flt-1 improves the chondrogenic potential of mouse skeletal muscle-derived stem cells.Terminal differentiation is not a major determinant for the success of stem cell therapy - cross-talk between muscle-derived stem cells and host cells.Pathogenetic mechanisms of posttraumatic osteoarthritis: opportunities for early intervention A Tunable Silk Hydrogel Device for Studying Limb Regeneration in Adult Xenopus Laevis.Hypoxia inducible factor (HIF) in rheumatology: low O2! See what HIF can do!Injecting vascular endothelial growth factor into the temporomandibular joint induces osteoarthritis in mice.CTGF increases vascular endothelial growth factor-dependent angiogenesis in human synovial fibroblasts by increasing miR-210 expression.Characterization of degenerative human facet joints and facet joint capsular tissues New findings in osteoarthritis pathogenesis: therapeutic implications.Targeting VEGF and Its Receptors for the Treatment of Osteoarthritis and Associated Pain.Oxygen and reactive oxygen species in articular cartilage: modulators of ionic homeostasis.Exercise-induced stem cell activation and its implication for cardiovascular and skeletal muscle regeneration.Amount of torque and duration of stretching affects correction of knee contracture in a rat model of spinal cord injury.Cartilage homeostasis in health and rheumatic diseases.Expression of hypoxia-inducible factor-1α in synovial fluid and articular cartilage is associated with disease severity in knee osteoarthritis.Cyr61 mediates the expression of VEGF, alphav-integrin, and alpha-actin genes through cytoskeletally based mechanotransduction mechanisms in bladder smooth muscle cells.Differential gene expression of human chondrocytes cultured under short-term altered gravity conditions during parabolic flight maneuvers.Undenatured type II collagen (UC-II®) for joint support: a randomized, double-blind, placebo-controlled study in healthy volunteers.TLR-2-mediated induction of vascular endothelial growth factor (VEGF) in cartilage in septic joint disease.Inhibition of vascular endothelial growth factor with shRNA in chondrocytes ameliorates osteoarthritis.Adeno-associated viral vector-mediated hypoxia-regulated VEGF gene transfer promotes angiogenesis following focal cerebral ischemia in mice.Pharmacological treatment with diacerein combined with mechanical stimulation affects the expression of growth factors in human chondrocytes.Action mechanisms of du-huo-ji-sheng-tang on cartilage degradation in a rabbit model of osteoarthritis.Vascular Endothelial Growth Factor in Cartilage Development and Osteoarthritis.The role of HIF-1alpha in maintaining cartilage homeostasis and during the pathogenesis of osteoarthritis.Sphingosine-1-phosphate modulates expression of vascular endothelial growth factor in human articular chondrocytes: a possible new role in arthritis.Estrogen modulates iodoacetate-induced gene expression in bovine cartilage explants.Biomaterial scaffolds in cartilage-subchondral bone defects influencing the repair of autologous articular cartilage transplants.Influence of corticosteroids on interleukin-1β-stimulated equine chondrocyte gene expression.

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P2860

Mechanical overload induces VEGF in cartilage discs via hypoxia-inducible factor.

http://www.wikidata.org/entity/Q38584496

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name

Mechanical overload induces VEGF in cartilage discs via hypoxia-inducible factor.

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Mechanical overload induces VEGF in cartilage discs via hypoxia-inducible factor.

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Mechanical overload induces VEGF in cartilage discs via hypoxia-inducible factor.

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The American Journal of Pathology

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Mechanical overload induces VEGF in cartilage discs via hypoxia-inducible factor.

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Alan J Grodzinsky

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Angelika Lemke

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Bernhard Tillmann

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Bodo Kurz

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Rolf Mentlein

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Thomas Pufe

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Wolf Petersen

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P2860

Activation of vascular endothelial growth factor gene transcription by hypoxia-inducible factor 1

Injurious mechanical compression of bovine articular cartilage induces chondrocyte apoptosis

The splice variants of vascular endothelial growth factor (VEGF) and their receptors

Biosynthetic response and mechanical properties of articular cartilage after injurious compression

Design and structure-activity relationship of a new class of potent VEGF receptor tyrosine kinase inhibitors

Vascular endothelial growth factor (VEGF) and its receptors

Regulation of translation initiation by FRAP/mTOR

Molecular and biological properties of vascular endothelial growth factor.

Osteoarthritis and cartilage: the role of cytokines.

Molecular mechanisms of blood vessel growth.

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