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Impact of hypokalemia on electromechanical window, excitation wavelength and repolarization gradients in guinea-pig and rabbit heartsRat strain-related differences in myocardial adrenergic tone and the impact on cardiac fibrosis, adrenergic responsiveness and myocardial structure and function.Activation of big conductance Ca(2+)-activated K (+) channels (BK) protects the heart against ischemia-reperfusion injury.Myocardial phosphodiesterases and regulation of cardiac contractility in health and cardiac disease.Cardiac hypertrophy induced by sustained beta-adrenoreceptor activation: pathophysiological aspects.Mechanisms of hypokalemia-induced ventricular arrhythmogenicity.Emerging role of neurotensin in regulation of the cardiovascular system.Susceptibility to systolic dysfunction in the myocardium from chronically infarcted spontaneously hypertensive rats.Chronic beta-adrenoreceptor activation increases cardiac cavity size through chamber remodeling and not via modifications in myocardial material properties.Inotropic responses to phosphodiesterase inhibitors in cardiac hypertrophy in rats.Electrophysiological determinants of arrhythmic susceptibility upon endocardial and epicardial pacing in guinea-pig heart.Reduced intrinsic heart rate is associated with reduced arrhythmic susceptibility in guinea-pig heart.Effects of Na+ channel blockers on extrasystolic stimulation-evoked changes in ventricular conduction and repolarization.Flecainide-induced proarrhythmia is attributed to abnormal changes in repolarization and refractoriness in perfused guinea-pig heart.Dofetilide promotes repolarization abnormalities in perfused Guinea-pig heart.Aldosterone receptor blockade prevents the transition to cardiac pump dysfunction induced by beta-adrenoreceptor activation.Neurotensin-induced myocardial noradrenergic effects in spontaneously hypertensive rats.Effects of ventricular pacing protocol on electrical restitution assessments in guinea-pig heart.Predictive value of electrical restitution in hypokalemia-induced ventricular arrhythmogenicity.Mechanisms of preserved baseline cardiac systolic function in rats with adrenergic inotropic downregulation.Impact and mechanisms of action of neurotensin on cardiac contractility in the rat left ventricle.Phosphodiesterase inhibition promotes the transition from compensated hypertrophy to cardiac dilatation in rats.Contractile responses to selective phosphodiesterase inhibitors following chronic beta-adrenoreceptor activation.Quinidine elicits proarrhythmic changes in ventricular repolarization and refractoriness in guinea-pig.Subtype-selective blockade of cardiac muscarinic receptors inhibits vagal chronotropic responses in cats.Procainamide and lidocaine produce dissimilar changes in ventricular repolarization and arrhythmogenicity in guinea-pig.Arrhythmogenic drugs can amplify spatial heterogeneities in the electrical restitution in perfused guinea-pig heart: An evidence from assessments of monophasic action potential durations and JT intervals.Determinants of slowed conduction in premature ventricular beats induced during programmed stimulations in perfused guinea-pig heartImpaired epicardial activation-repolarization coupling contributes to the proarrhythmic effects of hypokalaemia and dofetilide in guinea pig ventriclesImpact of Na⁺ channel blockers on transmural dispersion of refractoriness and arrhythmic susceptibility in guinea-pig left ventricleChronic sympathetic activation promotes downregulation of β-adrenoceptor-mediated effects in the guinea pig heart independently of structural remodeling and systolic dysfunctionMyocardial structural, contractile and electrophysiological changes in the guinea-pig heart failure model induced by chronic sympathetic activationNa+ channel distribution and electrophysiological heterogeneities in guinea pig ventricular wallElectrophysiological determinants of hypokalaemia-induced arrhythmogenicity in the guinea-pig heartChamber-specific effects of hypokalaemia on ventricular arrhythmogenicity in isolated, perfused guinea-pig heartCardiotropic effects of Met-enkephalin and somatostatin under conditions of neurotensin receptor blockadeImpact of chronic beta-adrenoceptor activation on neurotensin-induced myocardial effects in ratsCardiac dilatation and pump dysfunction without intrinsic myocardial systolic failure following chronic beta-adrenoreceptor activationTemporal changes in myocardial adrenergic regulation with the progression to pump dysfunction after chronic beta-adrenoreceptor activation in ratsAntiarrhythmic drug effects on premature beats are partly determined by prior cardiac activation frequency in perfused guinea-pig heart
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hulumtues
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հետազոտող
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Oleg E. Osadchii
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Oleg E. Osadchii
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Oleg E. Osadchii
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Oleg E. Osadchii
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Oleg E. Osadchii
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Oleg E. Osadchii
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Oleg E. Osadchii
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Oleg E. Osadchii
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Oleg E. Osadchii
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Oleg E. Osadchii
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Oleg E. Osadchii
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Oleg E. Osadchii
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Oleg E. Osadchii
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P106
P1153
7004509200
P21
P31
P496
0000-0003-0222-3227