Models of drug-induced epileptiform synchronization in vitro.
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Specific imbalance of excitatory/inhibitory signaling establishes seizure onset pattern in temporal lobe epilepsy.Dl-3n-butylphthalide reduces epileptiform activity through GluA2-lacking calcium-permeable AMPARs in epilepsy models.Excitatory Synaptic Input to Hilar Mossy Cells under Basal and Hyperexcitable Conditions.BAD and KATP channels regulate neuron excitability and epileptiform activity.Intrinsic Inflammation Is a Potential Anti-Epileptogenic Target in the Organotypic Hippocampal Slice Model.Potassium Channel Gain of Function in Epilepsy: An Unresolved Paradox.The combination of ribose and adenine promotes adenosine release and attenuates the intensity and frequency of epileptiform activity in hippocampal slices: Evidence for the rapid depletion of cellular ATP during electrographic seizures
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P2860
Models of drug-induced epileptiform synchronization in vitro.
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2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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name
Models of drug-induced epileptiform synchronization in vitro.
@en
type
label
Models of drug-induced epileptiform synchronization in vitro.
@en
prefLabel
Models of drug-induced epileptiform synchronization in vitro.
@en
P2860
P1476
Models of drug-induced epileptiform synchronization in vitro.
@en
P2093
John G R Jefferys
Massimo Avoli
P2860
P356
10.1016/J.JNEUMETH.2015.10.006
P577
2015-10-17T00:00:00Z