Innate Immune Activation Can Trigger Experimental Spondyloarthritis in HLA-B27/Huβ2m Transgenic Rats.

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Innate Immune Activation Can Trigger Experimental Spondyloarthritis in HLA-B27/Huβ2m Transgenic Rats.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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name

Innate Immune Activation Can T ...... HLA-B27/Huβ2m Transgenic Rats.

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label

Innate Immune Activation Can T ...... HLA-B27/Huβ2m Transgenic Rats.

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Innate Immune Activation Can T ...... HLA-B27/Huβ2m Transgenic Rats.

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Innate Immune Activation Can T ...... HLA-B27/Huβ2m Transgenic Rats.

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Desirée Pots

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Dominique L Baeten

http://www.w3.org/2001/XMLSchema#string

Gleb Slobodin

http://www.w3.org/2001/XMLSchema#string

Joel D Taurog

http://www.w3.org/2001/XMLSchema#string

Leonie M van Duivenvoorde

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Marleen G van de Sande

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Martha Dorris

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Melissa N van Tok

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Nimman Satumtira

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P2860

A proposed classification of the immunological diseases

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Enteric pathogens and reactive arthritis: a systematic review of Campylobacter, salmonella and Shigella-associated reactive arthritis

From HLA-B27 to spondyloarthritis: a journey through the ER.

Autoimmune epididymoorchitis is essential to the pathogenesis of male-specific spondylarthritis in HLA-B27-transgenic rats

Therapeutic and prophylactic thalidomide in TNBS-induced colitis: synergistic effects on TNF-alpha, IL-12 and VEGF production.

HLA-B27.

Arthritis induced in rats with nonimmunogenic adjuvants as models for rheumatoid arthritis.

Th17 cells expressing KIR3DL2+ and responsive to HLA-B27 homodimers are increased in ankylosing spondylitis.

Ankylosing spondylitis macrophage production of higher levels of interleukin-23 in response to lipopolysaccharide without induction of a significant unfolded protein response.

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scholarly article

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10.3389/FIMMU.2017.00920

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2017-08-07T00:00:00Z

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28824645

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5545590

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