Trigeminovascular calcitonin gene-related peptide function in Cacna1a R192Q-mutated knock-in mice.

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Trigeminovascular calcitonin gene-related peptide function in Cacna1a R192Q-mutated knock-in mice.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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Trigeminovascular calcitonin g ...... a R192Q-mutated knock-in mice.

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Trigeminovascular calcitonin g ...... a R192Q-mutated knock-in mice.

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Trigeminovascular calcitonin g ...... a R192Q-mutated knock-in mice.

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P1476

Trigeminovascular calcitonin g ...... 1a R192Q-mutated knock-in mice

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P2093

Antoinette MaassenVanDenBrink

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Arn Mjm van den Maagdenberg

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Ingrid M Garrelds

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Kayi Y Chan

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Martha B Ramírez-Rosas

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Sieneke Labruijere

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P2860

A Cacna1a knockin migraine mouse model with increased susceptibility to cortical spreading depression

Familial hemiplegic migraine and episodic ataxia type-2 are caused by mutations in the Ca2+ channel gene CACNL1A4

alpha-eudesmol, a P/Q-type Ca(2+) channel blocker, inhibits neurogenic vasodilation and extravasation following electrical stimulation of trigeminal ganglion.

Effects of ionotropic glutamate receptor antagonists on rat dural artery diameter in an intravital microscopy model.

The mechanism of functional up-regulation of P2X3 receptors of trigeminal sensory neurons in a genetic mouse model of familial hemiplegic migraine type 1 (FHM-1).

Pathophysiology of the migraine aura. The spreading depression theory.

Activity-dependent calcium, oxygen, and vascular responses in a mouse model of familial hemiplegic migraine type 1.

Regulation of smooth muscle calcium sensitivity: KCl as a calcium-sensitizing stimulus.

Migraine: gene mutations and functional consequences.

Cardiovascular responses produced by 5-hydroxytriptamine:a pharmacological update on the receptors/mechanisms involved and therapeutic implications.

P304

718-729

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scholarly article

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10.1177/0271678X17725673

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P433

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P478

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Alejandro Labastida-Ramírez

Alexander Hj Danser

P577

2017-08-09T00:00:00Z

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P698

28792272

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P932

6446415

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