JAK1/STAT3 activation through a proinflammatory cytokine pathway leads to resistance to molecularly targeted therapy in non-small cell lung cancer.
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NFAT1-regulated IL6 signalling contributes to aggressive phenotypes of glioma.Targeted inhibition of STAT/TET1 axis as a therapeutic strategy for acute myeloid leukemia.Amelioration of Experimental autoimmune encephalomyelitis and DSS induced colitis by NTG-A-009 through the inhibition of Th1 and Th17 cells differentiation.Overcoming EGFR-mediated osimertinib resistance through unique binding characteristics of second-generation EGFR inhibitors
P2860
JAK1/STAT3 activation through a proinflammatory cytokine pathway leads to resistance to molecularly targeted therapy in non-small cell lung cancer.
description
2017 nî lūn-bûn
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2017年の論文
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2017年論文
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2017年論文
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2017年論文
@zh-hk
2017年論文
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2017年論文
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2017年论文
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2017年论文
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2017年论文
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name
JAK1/STAT3 activation through ...... in non-small cell lung cancer.
@en
type
label
JAK1/STAT3 activation through ...... in non-small cell lung cancer.
@en
prefLabel
JAK1/STAT3 activation through ...... in non-small cell lung cancer.
@en
P2093
P2860
P50
P1476
JAK1/STAT3 activation through ...... in non-small cell lung cancer.
@en
P2093
Carmen Behrens
Dennis Ruder
J Jack Lee
John D Minna
Jonathan M Kurie
Juhee Song
Julie G Izzo
Kazuhiko Shien
P2860
P356
10.1158/1535-7163.MCT-17-0148
P577
2017-07-20T00:00:00Z